Donckerwolcke R A, Valk C, van Wijngaarden-Penterman M J, van Stekelenburg G J
Pediatr Res. 1979 Oct;13(10):1177-8. doi: 10.1203/00006450-197910000-00020.
The diagnosis of transient renal tubular acidosis was made in a 16 months old boy. Bicarbonate titration studies revealed that the acidification defect consisted of an association of proximal and distal tubular acidosis. The effect of experimentally induced potassium depletion revealed that hyperkalemia contributed to the acidification defect. After correction of the acidification disorder a defect in urinary concentration was still present.
一名16个月大的男孩被诊断为暂时性肾小管酸中毒。碳酸氢盐滴定研究表明,酸化缺陷由近端和远端肾小管酸中毒共同组成。实验性诱导钾缺乏的影响显示,高钾血症导致了酸化缺陷。在纠正酸化障碍后,尿浓缩功能仍存在缺陷。