培养的黑质外植体中的多巴胺能神经元。 - Suppr

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超能文献

Dopaminergic neurons in explants of substantia nigra in culture.

作者信息

Coyle J T, Jacobowitz D, Klein D, Axelrod J

出版信息

J Neurobiol. 1973;4(5):461-70. doi: 10.1002/neu.480040507.

DOI:10.1002/neu.480040507

PMID:4147763

Abstract

摘要

相似文献

Dopaminergic neurons in explants of substantia nigra in culture.培养的黑质外植体中的多巴胺能神经元。

J Neurobiol. 1973;4(5):461-70. doi: 10.1002/neu.480040507.

Mesencephalic neural stem (progenitor) cells develop to dopaminergic neurons more strongly in dopamine-depleted striatum than in intact striatum.中脑神经干细胞（祖细胞）在多巴胺耗尽的纹状体中比在完整纹状体中更易发育为多巴胺能神经元。

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Long-term glial cell line-derived neurotrophic factor overexpression in the intact nigrostriatal system in rats leads to a decrease of dopamine and increase of tetrahydrobiopterin production.长期在大鼠完整的黑质纹状体系统中过表达胶质细胞源性神经营养因子会导致多巴胺减少以及四氢生物蝶呤生成增加。

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Sequential administration of GDNF into the substantia nigra and striatum promotes dopamine neuron survival and axonal sprouting but not striatal reinnervation or functional recovery in the partial 6-OHDA lesion model.在部分6-羟基多巴胺损伤模型中，将胶质细胞源性神经营养因子（GDNF）依次注入黑质和纹状体可促进多巴胺能神经元存活和轴突发芽，但不能促进纹状体再支配或功能恢复。

Exp Neurol. 2000 Feb;161(2):503-16. doi: 10.1006/exnr.1999.7296.

Mesencephalic dopaminergic neurons protected by GDNF from axotomy-induced degeneration in the adult brain.在成体大脑中，中脑多巴胺能神经元受胶质细胞源性神经营养因子保护，免受轴突切断诱导的退化影响。

Nature. 1995 Jan 26;373(6512):339-41. doi: 10.1038/373339a0.

Delivery of a GDNF gene into the substantia nigra after a progressive 6-OHDA lesion maintains functional nigrostriatal connections.在进行性6-羟多巴胺损伤后，将胶质细胞源性神经营养因子（GDNF）基因导入黑质可维持功能性黑质纹状体连接。

Exp Neurol. 2000 Nov;166(1):1-15. doi: 10.1006/exnr.2000.7463.

Striatal GDNF administration increases tyrosine hydroxylase phosphorylation in the rat striatum and substantia nigra.纹状体内给予胶质细胞源性神经营因子可增加大鼠纹状体和黑质中酪氨酸羟化酶的磷酸化水平。

J Neurochem. 2004 Jul;90(1):245-54. doi: 10.1111/j.1471-4159.2004.02496.x.

Glial cell line-derived neurotrophic factor (GDNF) gene delivery protects dopaminergic terminals from degeneration.胶质细胞系源性神经营养因子（GDNF）基因传递可保护多巴胺能终末免于退化。

Exp Neurol. 2001 May;169(1):83-95. doi: 10.1006/exnr.2001.7638.

The antioxidant drink effective microorganism-X (EM-X) pre-treatment attenuates the loss of nigrostriatal dopaminergic neurons in 6-hydroxydopamine-lesion rat model of Parkinson's disease.抗氧化剂饮料有效微生物-X（EM-X）预处理可减轻帕金森病6-羟基多巴胺损伤大鼠模型中黑质纹状体多巴胺能神经元的损失。

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Cyclosporin A attenuates the decrease in tyrosine hydroxylase immunoreactivity in nigrostriatal dopaminergic neurons and in striatal dopamine content in rats with intrastriatal injection of 6-hydroxydopamine.环孢菌素A可减轻纹状体内注射6-羟基多巴胺的大鼠黑质纹状体多巴胺能神经元中酪氨酸羟化酶免疫反应性及纹状体多巴胺含量的降低。

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引用本文的文献

Ultrastructural localization of acetylcholinesterase activity in primary cultures of rat substantia nigra.大鼠黑质原代培养物中乙酰胆碱酯酶活性的超微结构定位

Histochemistry. 1980;70(1):7-18. doi: 10.1007/BF00508840.

Demonstration of enkephalin-, substance P- and glutamate decarboxylase-like immunoreactivity in cultured cells derived from newborn rat neostriatum.新生大鼠新纹状体来源的培养细胞中脑啡肽、P物质和谷氨酸脱羧酶样免疫反应性的显示

Histochemistry. 1980;69(2):169-79. doi: 10.1007/BF00533134.

Rat ventral mesencephalon grown as organotypic slice cultures and co-cultured with striatum, hippocampus, and cerebellum.大鼠腹侧中脑作为器官型切片培养物生长，并与纹状体、海马体和小脑共同培养。

Exp Brain Res. 1990;82(3):547-65. doi: 10.1007/BF00228796.

Quantitation of fiber growth in transplanted central monoamine neurons.

Cell Tissue Res. 1977 Apr 20;179(3):285-316. doi: 10.1007/BF00221102.

Explant cultures of catecholamine-containing neurons from rat brain: biochemical, histofluorescence, and electron microscopic studies.大鼠脑含儿茶酚胺神经元的组织块培养：生化、组织荧光及电子显微镜研究

Proc Natl Acad Sci U S A. 1977 Oct;74(10):4471-5. doi: 10.1073/pnas.74.10.4471.

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