免疫溶解转化：豚鼠补体系统中第八成分反应产生的一种不可逆损伤状态。 - Suppr

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超能文献

Immune lytic transformation: a state of irreversible damage generated as a result of the reaction of the eighth component in the guinea pig complement system.

作者信息

Stolfi R L

出版信息

J Immunol. 1968 Jan;100(1):46-54.

PMID:4169697

Abstract

摘要

相似文献

Immune lytic transformation: a state of irreversible damage generated as a result of the reaction of the eighth component in the guinea pig complement system.免疫溶解转化：豚鼠补体系统中第八成分反应产生的一种不可逆损伤状态。

J Immunol. 1968 Jan;100(1):46-54.

Immune hemolysis: a simplified method for the preparation of EAC'4 with guinea pig or with human complement.免疫溶血：一种用豚鼠或人补体制备EAC'4的简化方法。

J Immunol. 1967 Aug;99(2):263-8.

Inhibition of the eighth component of complement (C8) by ethylenediaminetetra-acetate (EDTA).乙二胺四乙酸（EDTA）对补体第八成分（C8）的抑制作用。

J Immunol. 1970 Feb;104(2):279-88.

Multiple nature of the third component of guinea-pig complement. II. Separation and description of two additional factors beta and d; preparation and characterization of four intermediate products.豚鼠补体第三成分的多重性质。II. 另外两个因子β和δ的分离与描述；四种中间产物的制备与特性分析

Immunology. 1965 Jun;8(6):604-17.

A specific method for purification of the second component of guinea pig complement and a chemical evaluation of the one-hit theory.豚鼠补体第二成分的一种纯化方法及“单次打击”理论的化学评估

J Immunol. 1970 Aug;105(2):327-41.

Multiple nature of the third component of guinea-pig complement. I. Separation and characterization of three factors a, b, and c, essential for haemolysis.豚鼠补体第三成分的多重性质。I. 溶血所必需的三种因子a、b和c的分离与特性鉴定。

Immunology. 1965 Jun;8(6):590-603.

The stoichiometric measurement of the serum inhibition of the first component of complement by the inhibition of immune hemolysis.通过免疫溶血抑制来进行血清对补体第一成分抑制作用的化学计量测量。

J Immunol. 1968 Jun;100(6):1154-64.

Complement: optimal reaction conditions for guinea pig C4.补体：豚鼠C4的最佳反应条件

J Immunol. 1973 Jul;111(1):189-99.

Molecular analysis of the reaction of C9 with EAC1-8: reaction of C9 with EAC1-8.C9与EAC1 - 8反应的分子分析：C9与EAC1 - 8的反应

J Immunol. 1974 Dec;113(6):1992-2003.

Canine hemolytic complement: optimal conditions for its titration.犬溶血补体：其滴定的最佳条件

Am J Vet Res. 1973 May;34(5):653-7.

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Glutathione inhibits antibody and complement-mediated immunologic cell injury via multiple mechanisms.谷胱甘肽通过多种机制抑制抗体和补体介导的免疫细胞损伤。

Redox Biol. 2017 Aug;12:571-581. doi: 10.1016/j.redox.2017.03.030. Epub 2017 Mar 31.

Genetic Association of the Porcine C9 Complement Component with Hemolytic Complement Activity.猪C9补体成分与溶血补体活性的遗传关联。

Asian-Australas J Anim Sci. 2015 Sep;28(9):1354-61. doi: 10.5713/ajas.14.0734.

An abnormal but functionally active complement component C9 protein found in an Irish family with subtotal C9 deficiency.在一个C9部分缺乏的爱尔兰家族中发现的一种异常但功能活跃的补体成分C9蛋白。

Immunology. 2003 Mar;108(3):384-90. doi: 10.1046/j.1365-2567.2003.01587.x.

Membrane attack complex of complement: a structural analysis of its assembly.补体膜攻击复合物：其组装的结构分析

J Exp Med. 1980 Feb 1;151(2):301-13. doi: 10.1084/jem.151.2.301.

Polymerization of the ninth component of complement (C9): formation of poly(C9) with a tubular ultrastructure resembling the membrane attack complex of complement.补体第九成分（C9）的聚合：形成具有类似于补体膜攻击复合物的管状超微结构的多聚（C9）。

Proc Natl Acad Sci U S A. 1982 Jan;79(2):574-8. doi: 10.1073/pnas.79.2.574.

The membrane attack complex.膜攻击复合物

Springer Semin Immunopathol. 1984;7(2-3):93-141. doi: 10.1007/BF01893017.

Is the membrane attack complex of complement an enzyme?补体的膜攻击复合物是一种酶吗？

Mol Cell Biochem. 1984;61(1):5-15. doi: 10.1007/BF00239603.

Single channel currents induced by complement in antibody-coated cell membranes.补体在抗体包被的细胞膜上诱导产生的单通道电流。

Proc Natl Acad Sci U S A. 1981 Oct;78(10):6421-5. doi: 10.1073/pnas.78.10.6421.

Direct measurement of the increase in intracellular free calcium ion concentration in response to the action of complement.直接测量细胞内游离钙离子浓度因补体作用而增加的情况。

Biochem J. 1981 Feb 15;194(2):551-60. doi: 10.1042/bj1940551.

Molecular organization of C9 within the membrane attack complex of complement. Induction of circular C9 polymerization by the C5b-8 assembly.补体膜攻击复合物中C9的分子组织。C5b-8组装诱导C9环状聚合。

J Exp Med. 1982 Jul 1;156(1):268-82. doi: 10.1084/jem.156.1.268.

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