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Hippocampal lesions produced by prolonged seizures in paralyzed artificially ventilated baboons.

作者信息

Meldrum B S, Vigouroux R A, Rage P, Brierley J B

出版信息

Experientia. 1973 May 15;29(5):561-3. doi: 10.1007/BF01926665.

DOI:10.1007/BF01926665
PMID:4199751
Abstract
摘要

相似文献

1
Hippocampal lesions produced by prolonged seizures in paralyzed artificially ventilated baboons.在人工通气的麻痹狒狒中,长时间癫痫发作所导致的海马损伤。
Experientia. 1973 May 15;29(5):561-3. doi: 10.1007/BF01926665.
2
Systemic factors and epileptic brain damage. Prolonged seizures in paralyzed, artificially ventilated baboons.全身因素与癫痫性脑损伤。瘫痪、人工通气的狒狒的长时间癫痫发作。
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Prolonged epileptic seizures in primates. Ischemic cell change and its relation to ictal physiological events.灵长类动物的长时间癫痫发作。缺血性细胞变化及其与发作期生理事件的关系。
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Four models for studying cerebral lesions secondary to epileptic seizures.四种用于研究癫痫发作继发脑损伤的模型。
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Physiological changes during prolonged seizures and epileptic brain damage.长时间癫痫发作及癫痫性脑损伤期间的生理变化。
Neuropadiatrie. 1978 Aug;9(3):203-12. doi: 10.1055/s-0028-1091481.
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Convulsive effects of bicuculline in photosensitive baboons (Papio papio) and rhesus monkeys (Macaca mulatta).荷包牡丹碱对光敏狒狒(豚尾狒狒)和恒河猴的惊厥作用。
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Neuronal loss and gliosis in the hippocampus following repetitive epileptic seizures induced in adolescent baboons by allylglycine.烯丙基甘氨酸诱导青春期狒狒反复癫痫发作后海马中的神经元丢失和胶质增生。
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[Cortical recordings by means of microelectrodes in photosensitive Papio papio].[通过微电极对光敏狒狒进行皮层记录]
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Hypothermia during kainic acid-induced seizures reduces hippocampal lesions and cerebral nitric oxide production in immature rabbits.
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Cortical hemodynamics of electrographic status epilepticus in the critically ill.危重症患者脑电图癫痫持续状态的皮质血流动力学
Epilepsia. 2025 Mar;66(3):802-816. doi: 10.1111/epi.18224. Epub 2024 Dec 26.
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Interictal spikes, seizures and ictal cell death are not necessary for post-traumatic epileptogenesis in vitro.发作间期棘波、癫痫发作和发作期细胞死亡对于体外创伤后癫痫发生不是必需的。
Neurobiol Dis. 2012 Feb;45(2):774-85. doi: 10.1016/j.nbd.2011.11.001. Epub 2011 Nov 13.
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In vivo contributions of BH3-only proteins to neuronal death following seizures, ischemia, and traumatic brain injury.

本文引用的文献

1
ETIOLOGY AND PATHOGENESIS OF TEMPORAL LOBE EPILEPSY.颞叶癫痫的病因与发病机制
Arch Neurol. 1964 Mar;10:233-48. doi: 10.1001/archneur.1964.00460150003001.
2
Experimental psychomotor epilepsy in the cat; electro-clinical and anatomo-pathological correlations.猫的实验性精神运动性癫痫;电临床与解剖病理学相关性
J Neuropathol Exp Neurol. 1959 Apr;18(2):270-93. doi: 10.1097/00005072-195904000-00004.
3
Genetic and related aetiological factors in temporal lobe epilepsy. A review.
在癫痫发作、缺血和创伤性脑损伤后,BH3 仅蛋白对神经元死亡的体内贡献。
J Cereb Blood Flow Metab. 2011 May;31(5):1196-210. doi: 10.1038/jcbfm.2011.26. Epub 2011 Mar 2.
Epilepsia. 1971 Mar;12(1):13-31. doi: 10.1111/j.1528-1157.1971.tb03912.x.
4
Intracarotid air embolism in the baboon: effects on cerebral blood flow and the electroencephalogram.
Brain Res. 1971 Jan 22;25(2):301-15. doi: 10.1016/0006-8993(71)90440-9.
5
Physiology of status epilepticus in primates.灵长类动物癫痫持续状态的生理学
Arch Neurol. 1973 Jan;28(1):1-9. doi: 10.1001/archneur.1973.00490190019001.
6
Prolonged epileptic seizures in primates. Ischemic cell change and its relation to ictal physiological events.灵长类动物的长时间癫痫发作。缺血性细胞变化及其与发作期生理事件的关系。
Arch Neurol. 1973 Jan;28(1):10-7. doi: 10.1001/archneur.1973.00490190028002.