Zeis P M, Rao S, John E G, Aschinberg L C
Acta Paediatr Scand. 1979 Mar;68(2):287-9. doi: 10.1111/j.1651-2227.1979.tb05006.x.
An 11-month-old boy had an episode of generalized convulsions followed by a right peripheral facial palsy, which resolved gradually within 3 weeks. Three months later he had another similar episode of convulsions followed by a left peripheral facial palsy. On both occasions it was found that he had polycythaemia. A careful physical examination discovered that the child had severe hypertension. Extensive laboratory investigations did not reveal a cause for his hypertension. Haematologic investigations showed that the polycythaemia was due to a contracted plasma volume as a result of the hypertension. The peripheral facial palsy most probably was due to a blood clot in the facial canal, below the origin of the nerve to m. stapedius, as audiograms were normal and lacrimation preserved. Control of the hypertension resulted in resolution of the facial palsy within 4 weeks and normal haematocrit readings within 6 weeks. It should be stressed that every patient with peripheral facial palsy should be examined for hypertension.
一名11个月大的男孩出现了一次全身性惊厥发作,随后出现右侧周围性面瘫,在3周内逐渐恢复。3个月后,他又出现了一次类似的惊厥发作,随后是左侧周围性面瘫。两次发作时均发现他患有红细胞增多症。仔细的体格检查发现该患儿患有严重高血压。广泛的实验室检查未发现其高血压的病因。血液学检查表明,红细胞增多症是由于高血压导致血浆容量收缩所致。由于听力图正常且泪液分泌保留,周围性面瘫很可能是由于面神经管内、镫骨肌神经起始部下方的血凝块所致。控制高血压后,面瘫在4周内恢复,血细胞比容在6周内恢复正常。应该强调的是,每一位周围性面瘫患者都应检查是否患有高血压。