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室性早搏耦联间期传至异位室性心动过速:一种非典型双向性心动过速的机制

Transfer of coupling of premature ventricular contractions to an ectopic ventricular tachycardia: a mechanism for atypical bidirectional tachycardia.

作者信息

Klein G J, Wallace A G, Gallagher J J

出版信息

J Electrocardiol. 1979 Jan;12(1):65-8. doi: 10.1016/s0022-0736(79)80046-1.

Abstract

Bidirectional tachycardia was observed on Holter tracings recorded from a dog three days after the production of a small freeze lesion on the wall of the left ventricle. A bigeminal rhythm with fixed coupling of ventricular premature contractions was interrupted by a burst of ventricular tachycardia of different morphology. The ventricular premature contractions, originally coupled to the sinus beats, became coupled to the new ventricular tachycardia with an identical coupling interval. This resulted in a dibirectional tachycardia, created by the "transfer" of coupling of premature ventricular contractions to a separate ectopic ventricular tachycardia.

摘要

在左心室壁制造一个小的冷冻损伤三天后,从一只狗记录的动态心电图上观察到双向性心动过速。室性早搏呈二联律且固定配对,被一阵形态不同的室性心动过速打断。最初与窦性搏动配对的室性早搏,以相同的配对间期与新的室性心动过速配对。这导致了一种双向性心动过速,是由室性早搏的配对“转移”到另一个异位室性心动过速所产生的。

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