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[On optimizing a feedback mechanism for additional tumor damage following a previous massive-dose therapy. Final step in therapy with manipulated poisoning of lysis-enzymes from the lysosomes of primary destructed tumor cells].

作者信息

von Ardenne M, Chaplain R A, Rieger F

出版信息

Z Krebsforsch. 1969;72(3):258-83.

PMID:4241683
Abstract
摘要

相似文献

1
[On optimizing a feedback mechanism for additional tumor damage following a previous massive-dose therapy. Final step in therapy with manipulated poisoning of lysis-enzymes from the lysosomes of primary destructed tumor cells].[关于优化先前大剂量治疗后额外肿瘤损伤的反馈机制。原发性破坏肿瘤细胞溶酶体中裂解酶的可控中毒治疗的最后一步]
Z Krebsforsch. 1969;72(3):258-83.
2
[The effect of histones on tumor cells].
Tsitologiia. 1974 Apr;16(4):481-5.
3
[In vitro effect of antitumor preparations on the hydrolase activity of lysosomes of tumor cells].
Vopr Onkol. 1974;20(10):62-6.
4
[Effect of antineoplastic agents on the activity and molecular heterogeneity of lysosomal hydrolases in Ehrlich ascites tumor].
Dokl Akad Nauk SSSR. 1974 Aug 1;217(4):961-4.
5
The effect of cancer chemotherapy on lysosomal enzymic activities (acid phosphatase) of Ehrlich ascites carcinoma cells.
Morphol Embryol (Bucur). 1975;21(3):219-26.
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Lethal action of sugars on ascites tumor cells in vitro.
Nature. 1967 Jan 21;213(5073):308-9. doi: 10.1038/213308a0.
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Influence of pH on the effect of hexamethylenetetramine on the growth of Ehrlich ascites tumors.
Oncology. 1973;28(5):411-27. doi: 10.1159/000224844.
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[Ineffectiveness of a drug combination on Yoshida sarcoma and Walker carcinosarcoma 256 of the rat and on Ehrlich carcinoma of the mouse].
Arzneimittelforschung. 1972 Mar;22(3):575-7.
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[Experiments to the lysosomal cytolysis chain reaction of cancer cells damage (author's transl)].
Z Naturforsch C Biosci. 1974 Jul-Aug;29(7-8):425-32.
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Lonidamine cytotoxicity: involvement of the lysosomal compartment.
J Int Med Res. 2000 Nov-Dec;28(6):269-76. doi: 10.1177/147323000002800603.

引用本文的文献

1
[Augmented lysosomal chain-reaction cytolysis through the temporary increase of lysosome content in cancerous cells].
Naturwissenschaften. 1970 Jul;57(7):360-1. doi: 10.1007/BF01173122.
2
[The pH-controlled basic process of general autolysis following the onset of death].
Naturwissenschaften. 1970 Jan;57(1):43-4. doi: 10.1007/BF00593566.
3
[Significance of cell injury chain reactions within the scope of multistep cancer therapy].
Klin Wochenschr. 1969 Jun 15;47(12):647-51. doi: 10.1007/BF01884360.
4
[High per cent remission of a spontaneous mouse mammary carcinoma owing to multiple stage cancer therapy].
Naturwissenschaften. 1969 Sep;56(9):464. doi: 10.1007/BF00601076.

本文引用的文献

1
THE EFFECT OF GLUCOSE PRETREATMENT ON THE CARCINOSTATIC AND TOXIC ACTIVITIES OF SOME ALKYLATING AGENTS.葡萄糖预处理对某些烷化剂的抑癌及毒性活性的影响
Biochem Pharmacol. 1964 Mar;13:395-400. doi: 10.1016/0006-2952(64)90158-3.
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Tissue fractionation studies. 10. Influence of ischaemia on the state of some bound enzymes in rat liver.组织分级分离研究。10. 缺血对大鼠肝脏中某些结合酶状态的影响。
Biochem J. 1959 Dec;73(4):610-6. doi: 10.1042/bj0730610.
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[The cytolyzing effect of extracts from normal tissues on various tumors in the rat].
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Prophylaxis of chemically induced papillomas and carcinomas of mouse skin by vitamin A-acid.维生素A酸对化学诱导的小鼠皮肤乳头瘤和癌的预防作用
Experientia. 1972 Oct 15;28(10):1219-20. doi: 10.1007/BF01946181.
[正常组织提取物对大鼠各种肿瘤的细胞溶解作用]
Z Krebsforsch. 1959;63:28-56.
4
Tissue fractionation studies. 9. Enzymic release of bound hydrolases.组织分级分离研究。9. 结合水解酶的酶促释放。
Biochem J. 1959 Dec;73(4):604-9. doi: 10.1042/bj0730604.
5
[Introduction to the physiopathology of the lysosomes].
Brux Med. 1966 Nov 13;46(46):1087-94.
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[Continuous perfusion of autolysing rat liver].
Hoppe Seylers Z Physiol Chem. 1966;344(1):55-74.
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[The most important statements of the mathematical in-vivo theory of the fermentation metabolism of cancerous tumors. Weak in-vivo effects in cancerous tumors with a strong in-vitro enzyme inhibition].
Dtsch Gesundheitsw. 1966 Dec 22;21(51):2412-8.
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[Spontaneous remission of tumors following hyperthermia--a feedback process?].
Naturwissenschaften. 1965 Dec;52(23):645. doi: 10.1007/BF00637701.
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[Selective damage to cancer cells by protein denaturation].
Dtsch Gesundheitsw. 1968 Sep 5;23(36):1681-5 contd.
10
[Selective enhancement of radiotherapeutic effect by manipulated tumor overacidification and heating].
Naturwissenschaften. 1968 Sep;55(9):448. doi: 10.1007/BF00602674.