因睾丸17-酮类固醇还原酶缺陷导致的伴有男性乳房发育的家族性男性假两性畸形。I. 体内研究。 - Suppr

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超能文献

Familial male pseudohermaphroditism with gynecomastia due to a testicular 17-ketosteroid reductase defect. I. Studies in vivo.

作者信息

Saez J M, De Peretti E, Morera A M, David M, Bertrand J

出版信息

J Clin Endocrinol Metab. 1971 May;32(5):604-10. doi: 10.1210/jcem-32-5-604.

DOI:10.1210/jcem-32-5-604

PMID:4252809

Abstract

摘要

相似文献

Familial male pseudohermaphroditism with gynecomastia due to a testicular 17-ketosteroid reductase defect. I. Studies in vivo.因睾丸17-酮类固醇还原酶缺陷导致的伴有男性乳房发育的家族性男性假两性畸形。I. 体内研究。

J Clin Endocrinol Metab. 1971 May;32(5):604-10. doi: 10.1210/jcem-32-5-604.

Further in vivo studies in male pseudohermaphroditism with gynecomastia due to a testicular 17-ketosteroid reductase defect (compared to a case of testicular feminization).对因睾丸17-酮类固醇还原酶缺陷导致男性假两性畸形伴男性乳腺增生的进一步体内研究（与睾丸女性化病例相比）。

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Familial male pseudohermaphroditism without gynecomastia due to deficient testicular 17-ketosteroid reductase activity.由于睾丸17-酮类固醇还原酶活性缺乏导致的无男子女性型乳房的家族性男性假两性畸形。

N Engl J Med. 1974 Oct 31;291(18):938-44. doi: 10.1056/NEJM197410312911805.

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Children with male pseudohermaphroditism: endocrine and metabolic studies.男性假两性畸形患儿：内分泌与代谢研究

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[Familial male pseudohermaphrodism caused by testicular 17-ketosteroid-reductase deficiency].[睾丸17-酮类固醇还原酶缺乏所致的家族性男性假两性畸形]

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In vivo studies on testosterone metabolism by skin of normal males and patients with the syndrome of testicular feminization.正常男性和睾丸女性化综合征患者皮肤对睾酮代谢的体内研究。

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Testicular 17-ketosteroid reductase deficiency as a cause of male pseudohermaphroditism in 2 sibs.

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Horm Metab Res. 1976 Jul;8(4):307-10. doi: 10.1055/s-0028-1093642.

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