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Metabolic alterations in Yoshida ascites tumour cells caused by 6-aminonicotinamide and prevented by nicotinamide.

作者信息

Ofori-Nkansah N, von Bruchhausen F

出版信息

Naunyn Schmiedebergs Arch Pharmacol. 1972;272(2):156-68. doi: 10.1007/BF00508766.

DOI:10.1007/BF00508766
PMID:4258288
Abstract
摘要

相似文献

1
Metabolic alterations in Yoshida ascites tumour cells caused by 6-aminonicotinamide and prevented by nicotinamide.6-氨基烟酰胺引起吉田腹水瘤细胞的代谢改变,而烟酰胺可预防这种改变。
Naunyn Schmiedebergs Arch Pharmacol. 1972;272(2):156-68. doi: 10.1007/BF00508766.
2
Some metabolic and morphological alterations in Yoshida ascites tumour cells caused by 6-aminonicotinamide.6-氨基烟酰胺引起的吉田腹水瘤细胞的一些代谢和形态学改变。
Z Krebsforsch Klin Onkol Cancer Res Clin Oncol. 1972;77(1):64-76. doi: 10.1007/BF00284355.
3
[The impairment of glycogen metabolism in adipose tissue by 6-aminonicotinamide as influenced by the inhibition of phosphofructokinase].[6-氨基烟酰胺对脂肪组织糖原代谢的损害:受磷酸果糖激酶抑制的影响]
Naunyn Schmiedebergs Arch Pharmacol. 1972;272(2):143-55. doi: 10.1007/BF00508765.
4
Transformation of glucosamine to glycogen and lactate by ascites tumor cells.腹水肿瘤细胞将氨基葡萄糖转化为糖原和乳酸。
Biochim Biophys Acta. 1971 Jul 20;244(1):19-29. doi: 10.1016/0304-4165(71)90116-4.
5
Cerebral energy reserves and glycolysis in neural tissue of 6-aminonicotinamide-treated mice.6-氨基烟酰胺处理小鼠神经组织中的脑能量储备与糖酵解
J Neurobiol. 1974;5(5):379-92. doi: 10.1002/neu.480050502.
6
Effects of chlorambucil treatment on the rates of respiration and glycolysis in drug-sensitive and -resistant strains of the Yoshida ascites sarcoma.苯丁酸氮芥治疗对吉田腹水肉瘤药物敏感和耐药菌株呼吸速率及糖酵解速率的影响。
Biochem Pharmacol. 1970 Dec;19(12):2973-8. doi: 10.1016/0006-2952(70)90083-3.
7
Metabolic alterations in the axotomized superior cervical ganglion of the rat. I. Energy metabolism.大鼠颈上神经节切断术后的代谢改变。I. 能量代谢
Brain Res. 1974 Jan 4;65(1):127-39. doi: 10.1016/0006-8993(74)90340-0.
8
In vitro-in vivo studies on the susceptibility of the solid Yoshida sarcoma to drugs and hyperthermia (42 degrees).
Cancer Res. 1974 Jun;34(6):1263-74.
9
Pyrimidine metabolism in normal and tumor tissues.正常组织与肿瘤组织中的嘧啶代谢。
Gan. 1972 Dec;63(6):685-92.
10
Some effects of chlorambucil on the chromatin of Yoshida ascites sarcoma cells.
Cancer Res. 1973 Feb;33(2):389-93.

引用本文的文献

1
Some metabolic and morphological alterations in Yoshida ascites tumour cells caused by 6-aminonicotinamide.6-氨基烟酰胺引起的吉田腹水瘤细胞的一些代谢和形态学改变。
Z Krebsforsch Klin Onkol Cancer Res Clin Oncol. 1972;77(1):64-76. doi: 10.1007/BF00284355.

本文引用的文献

1
6-Aminonicotinamide--a Potent Nicotinamide Antagonist.6-氨基烟酰胺——一种强效烟酰胺拮抗剂。
Science. 1955 Oct 28;122(3174):834. doi: 10.1126/science.122.3174.834.
2
The kinetics of phosphoglucoisomerase.磷酸葡萄糖异构酶的动力学
J Biol Chem. 1960 Aug;235:2178-84.
3
SOME PROPERTIES OF THE ENZYME NICOTINAMIDE ADENINE DINUCLEOTIDE GLYCOHYDROLASE FROM MOUSE EHRLICH ASCITES CELLS.来自小鼠艾氏腹水癌细胞的烟酰胺腺嘌呤二核苷酸糖水解酶的一些特性
J Biol Chem. 1964 Aug;239:2613-7.
4
[INFLUENCE OF NADP ANALOGUES ON THE REACTION RATE OF SOME NADP-DEPENDENT OXIDOREDUCTASES].[烟酰胺腺嘌呤二核苷酸磷酸类似物对某些依赖烟酰胺腺嘌呤二核苷酸磷酸的氧化还原酶反应速率的影响]
Biochim Biophys Acta. 1964 Jul 8;89:23-32.
5
[BIOSYNTHESIS AND EFFECT OF 6-AMINONICOTINAMIDE-ADENINE DINUCLEOTIDE (6-ANAD)].6-氨基烟酰胺-腺嘌呤二核苷酸(6-ANAD)的生物合成及作用
Naunyn Schmiedebergs Arch Exp Pathol Pharmakol. 1964 Feb 13;246:437-51.
6
FORMATION OF GLYCOLYTIC INHIBITOR FROM 6-AMINONICOTINAMIDE BY ASCITES TUMOR CELLS IN VIVO AND IN VITRO, AND METABOLIC REQUIREMENTS FOR FORMATION OF THIS INHIBITOR.腹水肿瘤细胞在体内和体外由6-氨基烟酰胺形成糖酵解抑制剂及其形成该抑制剂的代谢需求
Biochem Z. 1963;338:381-92.
7
Clinical use of 6-aminonicotinamide in patients with disseminated neoplastic disease.6-氨基烟酰胺在播散性肿瘤疾病患者中的临床应用。
Cancer. 1961 May-Jun;14:644-8. doi: 10.1002/1097-0142(199005/06)14:3<644::aid-cncr2820140328>3.0.co;2-n.
8
Clinical experience with 6-aminonicotinamide.6-氨基烟酰胺的临床经验
Cancer Res. 1961 Jan;21:31-7.
9
Quantitative biochemical differences between tumor and host tissue. VI. 6-Aminonicotinamide antagonism of DPN-dependent enzymatic systems.
Cancer Res. 1958 Dec;18(11):1272-80.
10
Pyridine nucleotide metabolism: mechanism of action of the niacin antagonist, 6-aminonicotinamide.吡啶核苷酸代谢:烟酸拮抗剂6-氨基烟酰胺的作用机制
J Biol Chem. 1958 Oct;233(4):964-8.