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Cardiomyopathic Syrian hamster: a possible model of human disease.

作者信息

Gertz E W

出版信息

Prog Exp Tumor Res. 1972;16:242-60. doi: 10.1159/000393374.

DOI:10.1159/000393374
PMID:4261129
Abstract
摘要

相似文献

1
Cardiomyopathic Syrian hamster: a possible model of human disease.
Prog Exp Tumor Res. 1972;16:242-60. doi: 10.1159/000393374.
2
[Hypertrophy of the heart muscle in hereditary myopathy. Studies on the Syrian hamster in the electron microscopy picture].
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3
The significance of biochemical and structural changes in the development of the myocardiopathy of the Syrian hamster.
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The cardiomyopathy of the Syrian hamster (strain BIO 8262)--hypertrophic or dystrophic?
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5
Tension-frequency relationships in normal and cardiomyopathic dog and hamster myocardium.正常及心肌病犬和仓鼠心肌中的张力-频率关系。
J Mol Cell Cardiol. 1995 Jun;27(6):1251-61. doi: 10.1016/s0022-2828(05)82387-9.
6
Several biochemical characteristics of the cardiomyopathic Syrian hamster.
Recent Adv Stud Cardiac Struct Metab. 1972;1:235-42.
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Fragmented sarcoplasmic reticulum of the cardiomyopathic Syrian hamster: lipid composition, Ca++ transport, and Ca++-stimulated ATPase.
Recent Adv Stud Cardiac Struct Metab. 1974;4:541-50.
8
[Spontaneously cardiomyopathic mice, KK (author's transl)].自发性心肌病小鼠,KK(作者译)
Jikken Dobutsu. 1978 Apr;27(2):197-9.
9
Interrelationships between reparative processes in myocardium and the development of congestive heart failure.
Rev Can Biol. 1968 Mar;27(1):45-60.
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Altered subcellular Ca2+ regulation in papillary muscles from cardiomyopathic hamster hearts.
Am J Physiol. 1995 May;268(5 Pt 2):H1875-83. doi: 10.1152/ajpheart.1995.268.5.H1875.

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1
Progress in gene therapy of dystrophic heart disease.肌营养不良性心脏病的基因治疗进展。
Gene Ther. 2012 Jun;19(6):678-85. doi: 10.1038/gt.2012.10. Epub 2012 Feb 9.
2
Reduced Ca2+ transport across sarcolemma but enhanced spontaneous activity in cardiomyocytes isolated from left atrium-pulmonary veins tissue of myopathic hamster.从病态仓鼠的左心房-肺静脉组织中分离出的心肌细胞中,肌细胞膜上 Ca2+转运减少,但自发性活动增强。
J Biomed Sci. 2009 Dec 29;16(1):114. doi: 10.1186/1423-0127-16-114.
3
AlphaB-crystallin, a low-molecular-weight heat shock protein, acts as a regulator of platelet function.
αB-晶状体蛋白,一种低分子量热休克蛋白,充当血小板功能的调节剂。
Cell Stress Chaperones. 2001 Jan;6(1):21-8. doi: 10.1379/1466-1268(2001)006<0021:bcalmw>2.0.co;2.
4
Verapamil induced reduction of the myocardial beta-adrenoceptor density in BIO 14.6 cardiomyopathic Syrian hamsters.维拉帕米可降低BIO 14.6心肌病叙利亚仓鼠的心肌β-肾上腺素能受体密度。
Mol Cell Biochem. 1993 Apr 7;121(1):59-65. doi: 10.1007/BF00928700.
5
Intrinsic connective tissue abnormalities in the heart muscle of cardiomyopathic Syrian hamsters.患心肌病的叙利亚仓鼠心肌的内在结缔组织异常。
Am J Pathol. 1987 May;127(2):327-34.
6
Stimulatory guanine nucleotide-binding protein and adenylate cyclase activities in Bio 14.6 cardiomyopathic hamsters at the hypertrophic stage.肥厚期Bio 14.6心肌病仓鼠中刺激性鸟嘌呤核苷酸结合蛋白和腺苷酸环化酶活性
Mol Cell Biochem. 1992 Mar 4;110(1):83-90. doi: 10.1007/BF02385009.