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Myocardial oxygen consumption in experimental hypertrophy and congestive heart failure due to pressure overload.

作者信息

Gunning J F, Cooper G, Harrison C E, Coleman H N

出版信息

Am J Cardiol. 1973 Sep 20;32(4):427-36. doi: 10.1016/s0002-9149(73)80033-5.

DOI:10.1016/s0002-9149(73)80033-5
PMID:4269699
Abstract
摘要

相似文献

1
Myocardial oxygen consumption in experimental hypertrophy and congestive heart failure due to pressure overload.
Am J Cardiol. 1973 Sep 20;32(4):427-36. doi: 10.1016/s0002-9149(73)80033-5.
2
Myocardial oxygen consumption during experimental hypertrophy and congestive heart failure.实验性心肌肥厚和充血性心力衰竭时的心肌耗氧量
J Mol Cell Cardiol. 1973 Feb;5(1):25-38. doi: 10.1016/0022-2828(73)90033-3.
3
Normal myocardial function and energetics after reversing pressure-overload hypertrophy.逆转压力超负荷肥大后的正常心肌功能和能量代谢。
Am J Physiol. 1974 May;226(5):1158-65. doi: 10.1152/ajplegacy.1974.226.5.1158.
4
Recuperative potential of cardiac muscle following relief of pressure overload hypertrophy and right ventricular failure in the cat.猫压力超负荷肥大及右心室衰竭缓解后心肌的恢复潜力
Circ Res. 1977 Jan;40(1):41-9. doi: 10.1161/01.res.40.1.41.
5
Normal myocardial function and energetics in volume-overload hypertrophy in the cat.猫容量超负荷性肥大时的正常心肌功能与能量代谢
Circ Res. 1973 Feb;32(2):140-8. doi: 10.1161/01.res.32.2.140.
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Contractile state of cardiac muscle obtained from cats with experimentally produced ventricular hypertrophy and heart failure.从实验性诱导心室肥厚和心力衰竭的猫身上获取的心肌收缩状态。
Circ Res. 1967 Sep;21(3):341-54. doi: 10.1161/01.res.21.3.341.
7
Control of myocardial oxygen consumption: physiologic and clinical considerations.心肌氧消耗的控制：生理与临床考量
Am J Cardiol. 1971 Apr;27(4):416-32. doi: 10.1016/0002-9149(71)90439-5.
8
Recovery of myosin ATPase after relief of pressure-overload hypertrophy and failure.
Am J Physiol. 1978 Jun;234(6):H711-7. doi: 10.1152/ajpheart.1978.234.6.H711.
9
Mechanisms for the abnormal energetics of pressure-induced hypertrophy of cat myocardium.猫心肌压力诱导性肥大异常能量代谢的机制。
Circ Res. 1973 Aug;33(2):213-23. doi: 10.1161/01.res.33.2.213.
10
Induction of heart failure by minimally invasive aortic constriction in mice: reduced peroxisome proliferator-activated receptor γ coactivator levels and mitochondrial dysfunction.经微创主动脉缩窄诱导的小鼠心力衰竭：过氧化物酶体增殖物激活受体 γ 共激活因子水平降低和线粒体功能障碍。
J Thorac Cardiovasc Surg. 2011 Feb;141(2):492-500, 500.e1. doi: 10.1016/j.jtcvs.2010.03.029. Epub 2010 May 5.

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Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signaling.
线粒体氧化应激通过激活单磷酸腺苷激活的蛋白激酶-α信号通路损害冠状动脉侧支生长。
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Biochem J. 2011 Apr 15;435(2):519-28. doi: 10.1042/BJ20101390.
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Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.生物能量储备能力在应对4-羟基壬烯醛诱导的心肌细胞应激中的重要性。
Biochem J. 2009 Oct 23;424(1):99-107. doi: 10.1042/BJ20090934.
6
Rate of pyridine nucleotide oxidation and cytochrome oxidase interaction with intracellular oxygen in hearts from rats with compensated volume overload.代偿性容量超负荷大鼠心脏中吡啶核苷酸氧化速率及细胞色素氧化酶与细胞内氧的相互作用
Pflugers Arch. 1981 Dec;392(2):106-14. doi: 10.1007/BF00581257.
7
Positive correlation between aortic valve pressure gradient and mitochondrial respiratory chain capacity in hypertrophied human left ventricle.肥厚型人类左心室中主动脉瓣压力阶差与线粒体呼吸链容量之间的正相关关系。
Clin Investig. 1992 Oct;70(10):896-901. doi: 10.1007/BF00180435.
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Anesthesia for open-heart surgery: Its contribution to the care of the critically ill.心脏直视手术的麻醉：其对危重症患者护理的贡献。
Bull N Y Acad Med. 1975 Sep;51(8):930-46.
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[Value and limitations of coronary blood flow measurement in man (author's transl)].[人体冠状动脉血流测量的价值与局限性（作者译）]
Klin Wochenschr. 1975 Aug 1;53(15):691-707. doi: 10.1007/BF01468700.
10
Alterations of mitochondrial function as detected in left ventricular myocardium of rats with acute aortic constriction.急性主动脉缩窄大鼠左心室心肌中线粒体功能的改变。
Basic Res Cardiol. 1978 Nov-Dec;73(6):535-50. doi: 10.1007/BF01906792.