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Metabolism of tritiated vitamin D3 in familial vitamin D-resistant rickets with hypophosphatemia.

作者信息

DeLuca H F, Lund J, Rosenbloom A, Lobeck C C

出版信息

J Pediatr. 1967 May;70(5):828-32. doi: 10.1016/s0022-3476(67)80342-1.

DOI:10.1016/s0022-3476(67)80342-1
PMID:4290149
Abstract
摘要

相似文献

1
Metabolism of tritiated vitamin D3 in familial vitamin D-resistant rickets with hypophosphatemia.
J Pediatr. 1967 May;70(5):828-32. doi: 10.1016/s0022-3476(67)80342-1.
2
Metabolism of vitamin D3-3H in vitamin D-resistant rickets and familial hypophosphatemia.维生素D抵抗性佝偻病和家族性低磷血症中维生素D3-3H的代谢
J Clin Invest. 1967 Dec;46(12):1907-15. doi: 10.1172/JCI105680.
3
Metabolism and serum protein binding of 3H-vitamin D4 in vitamin D-resistant rickets.
J Pediatr. 1969 May;74(5):828. doi: 10.1016/s0022-3476(69)80171-x.
4
Effects of 25-hydroxycholecalciferol in patients with familial hypophosphatemia and vitamin-D-resistant rickets.
N Engl J Med. 1970 Sep 17;283(12):627-30. doi: 10.1056/NEJM197009172831204.
5
[Net intestinal absorption of 25-hydroxychlecalciferol in children].[儿童25-羟胆钙化醇的肠道净吸收]
C R Acad Hebd Seances Acad Sci D. 1972 Jun 5;274(23):3152-5.
6
The role of abnormal vitamin D metabolism in X-linked hypophosphatemic rickets and osteomalacia.
Adv Exp Med Biol. 1984;178:399-404. doi: 10.1007/978-1-4684-4808-5_48.
7
[X-linked familial hypophosphatemic rickets report of six cases (author's transl)].X连锁家族性低磷血症佝偻病六例报告（作者译）
An Esp Pediatr. 1979 Mar;12(3):189-98.
8
[Molecular mechanisms of familial rachitis].[家族性佝偻病的分子机制]
Arch Pediatr. 1997;4(2 Suppl 2):102s-105s. doi: 10.1016/s0929-693x(97)86472-x.
9
Familial hypophosphatemic rickets: a delineation of its subdivisions and pathogenesis.家族性低磷血症性佝偻病：其亚分类及发病机制的阐述
Clin Pediatr (Phila). 1976 Nov;15(11):1007-12. doi: 10.1177/000992287601501106.
10
[Vitamin D-resistant hypophosphatemic rickets].[维生素D抵抗性低磷血症性佝偻病]
Ugeskr Laeger. 1977 Jan;139(3):147-8.

引用本文的文献

1
Metabolites of vitamin D in normal and X-linked hypophosphatemic mice.正常和X连锁低磷血症小鼠体内维生素D的代谢产物
Calcif Tissue Int. 1984 Dec;36(6):662-7. doi: 10.1007/BF02405387.
2
Pathogenesis of rickets and osteomalacia in familial hypophosphataemia.家族性低磷血症性佝偻病和骨软化症的发病机制。
Arch Dis Child. 1971 Jun;46(247):269-72. doi: 10.1136/adc.46.247.269.
3
Metabolic bone disease secondary to renal and intestinal disorders.继发于肾脏和肠道疾病的代谢性骨病。
Calif Med. 1969 May;110(5):397-409.
4
25-Hydroxycholecalciferol. A comparative study in deficiency rickets and different types of resistant rickets.
J Clin Invest. 1972 Apr;51(4):749-59. doi: 10.1172/JCI106869.
5
Biological activity of 25-hydroxycholecalciferol, a metabolite of vitamin D3.维生素D3的代谢产物25-羟基胆钙化醇的生物活性。
Proc Natl Acad Sci U S A. 1968 Dec;61(4):1503-6. doi: 10.1073/pnas.61.4.1503.
6
The biological activity of 25-hydroxycholecalciferol, a metabolite of vitamin D3.维生素D3的代谢产物25-羟基胆钙化醇的生物活性。
Proc Natl Acad Sci U S A. 1968 Oct;61(2):717-8. doi: 10.1073/pnas.61.2.717.
7
Metabolism of vitamin D3-3H in vitamin D-resistant rickets and familial hypophosphatemia.维生素D抵抗性佝偻病和家族性低磷血症中维生素D3-3H的代谢
J Clin Invest. 1967 Dec;46(12):1907-15. doi: 10.1172/JCI105680.