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Hypernatremia due to hypodipsia and elevated threshold for vasopressin release. Effects of treatment with hydrochlorothiazide, chlorpropamide and tolbutamide.

作者信息

Mahoney J H, Goodman A D

出版信息

N Engl J Med. 1968 Nov 28;279(22):1191-6. doi: 10.1056/NEJM196811282792203.

DOI:10.1056/NEJM196811282792203
PMID:4301177
Abstract
摘要

相似文献

1
Hypernatremia due to hypodipsia and elevated threshold for vasopressin release. Effects of treatment with hydrochlorothiazide, chlorpropamide and tolbutamide.因渴感减退和抗利尿激素释放阈值升高导致的高钠血症。氢氯噻嗪、氯磺丙脲和甲苯磺丁脲治疗的效果。
N Engl J Med. 1968 Nov 28;279(22):1191-6. doi: 10.1056/NEJM196811282792203.
2
Successful treatment of hypernatremic thirst deficiency with chlorpropamide.用氯磺丙脲成功治疗高钠血症性渴觉缺乏症。
Clin Nephrol. 1978 Sep;10(3):90-5.
3
[Essential hypernatremia with hypovolemia caused by hypodipsia and partial diabetes insipidus].[因饮水过少和部分性尿崩症导致的伴有低血容量的原发性高钠血症]
Sem Hop. 1977 May 23;53(20):1137-42.
4
Syndrome of hypernatremia, hypodipsia and partial diabetes insipidus: a new interpretation.高钠血症、低渴感和部分性尿崩症综合征：一种新的解释。
J Clin Endocrinol Metab. 1974 May;38(5):890-901. doi: 10.1210/jcem-38-5-890.
5
[Chlorpropamide, tolbutamide and saluretics in the treatment of diabetes insipidus sensitive to vasopressin].[氯磺丙脲、甲苯磺丁脲及利尿药治疗血管加压素敏感型尿崩症]
Rev Clin Esp. 1970 Feb 15;116(3):237-42.
6
[Hypernatremia and adipsia (author's transl)].高钠血症与渴感缺乏（作者译）
An Esp Pediatr. 1979 Feb;12(2):137-44.
7
Restoration of normal drinking behavior by chlorpropamide in patients with hypodipsia and diabetes insipidus.氯磺丙脲对烦渴减退和尿崩症患者正常饮水行为的恢复作用
Am J Med. 1971 Sep;51(3):304-13. doi: 10.1016/0002-9343(71)90265-8.
8
Essential hypernatraemia, antidiuretic hormone and neurophysin secretion: response to chlorpropamide.原发性高钠血症、抗利尿激素与神经垂体素分泌：对氯磺丙脲的反应
Acta Endocrinol (Copenh). 1981 Feb;96(2):145-53. doi: 10.1530/acta.0.0960145.
9
Hypodipsia and hypernatraemia associated with hypothalamic and suprasellar lesions.与下丘脑和鞍上病变相关的渴感减退和高钠血症。
Brain. 1972;95(2):249-64. doi: 10.1093/brain/95.2.249.
10
[ADH-like mechanism of action of chlorpropamide in diabetes insipidus (a comparison with the antidiuretic effect of other blood glucose decreasing sulfonamides and hydrochlorothiazide)].[氯磺丙脲在尿崩症中的抗利尿激素样作用机制（与其他降血糖磺酰胺类药物及氢氯噻嗪的抗利尿作用比较）]
Klin Wochenschr. 1970 Jul 15;48(14):855-61. doi: 10.1007/BF01583906.

引用本文的文献

1
PROTRACTED ACUTE HYPERVOLEMIC HYPERNATREMIA UNMASKED AFTER VASOPRESSIN THERAPY: CASE REPORT, LITERATURE REVIEW, AND PROPOSED ALGORITHMIC APPROACH.血管加压素治疗后发现的迁延性急性高血容量性高钠血症：病例报告、文献综述及拟议的算法方法
AACE Clin Case Rep. 2018 Oct 5;5(2):e95-e98. doi: 10.4158/ACCR-2018-0363. eCollection 2019 Mar-Apr.
2
Hypernatremic thirst deficiency and memory disorders following hypothalamic lesions.下丘脑病变后的高钠血症性渴觉缺乏与记忆障碍
Arch Psychiatr Nervenkr (1970). 1982;231(5):459-70. doi: 10.1007/BF00342725.
3
Hereditary central diabetes insipidus: plasma levels of antidiuretic hormone in a family with a possible osmoreceptor defect.
遗传性中枢性尿崩症：一个可能存在渗透压感受器缺陷的家族中抗利尿激素的血浆水平
Can Med Assoc J. 1984 Nov 15;131(10):1237-41.
4
[The mode of action of chlorpropamide in diabetes insipidus].
Klin Wochenschr. 1971 Mar 15;49(6):314-22. doi: 10.1007/BF01496450.
5
[Diuretic and natriuretic action of chlorpropamide in rats. Comparison with tolbutamide].[氯磺丙脲对大鼠的利尿和利钠作用。与甲苯磺丁脲的比较]
Experientia. 1971 Feb 15;27(2):149-51. doi: 10.1007/BF02145862.
6
"Essential" hypernatremia due to ineffective osmotic and intact volume regulation of vasopressin secretion.由于血管加压素分泌的渗透调节无效和容量调节正常而导致的“原发性”高钠血症。
J Clin Invest. 1971 Jan;50(1):97-111. doi: 10.1172/JCI106489.
7
Chlorpropamide in diabetes insipidus.氯磺丙脲治疗尿崩症。
Br Med J. 1971 Feb 6;1(5744):302-3.
8
[ADH-like mechanism of action of chlorpropamide in diabetes insipidus (a comparison with the antidiuretic effect of other blood glucose decreasing sulfonamides and hydrochlorothiazide)].[氯磺丙脲在尿崩症中的抗利尿激素样作用机制（与其他降血糖磺酰胺类药物及氢氯噻嗪的抗利尿作用比较）]
Klin Wochenschr. 1970 Jul 15;48(14):855-61. doi: 10.1007/BF01583906.
9
Acute water intoxication from compulsive drinking.强迫性饮水导致的急性水中毒
Br Med J. 1973 May 12;2(5862):365. doi: 10.1136/bmj.2.5862.365-c.
10
Diabetes insipidus.尿崩症
Br Med J. 1972 Apr 22;2(5807):210-3. doi: 10.1136/bmj.2.5807.210.