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Biochemical changes in rat liver in response to treatment with drugs and other agents. II. Effects of halothane, DDT, other chlorinated hydrocarbons, thioacetamide, dimethylnitrosamine and ethionine.

作者信息

Platt D S, Cockrill B L

出版信息

Biochem Pharmacol. 1969 Feb;18(2):445-57. doi: 10.1016/0006-2952(69)90221-4.

DOI:10.1016/0006-2952(69)90221-4
PMID:4305095
Abstract
摘要

相似文献

1
Biochemical changes in rat liver in response to treatment with drugs and other agents. II. Effects of halothane, DDT, other chlorinated hydrocarbons, thioacetamide, dimethylnitrosamine and ethionine.大鼠肝脏对药物及其他制剂处理的生化变化。II. 氟烷、滴滴涕、其他氯代烃、硫代乙酰胺、二甲基亚硝胺和乙硫氨酸的影响。
Biochem Pharmacol. 1969 Feb;18(2):445-57. doi: 10.1016/0006-2952(69)90221-4.
2
Liver enlargement and hepatoxicity: an investigation into the effects of several agents on rat liver enzyme activities.
Biochem Pharmacol. 1967 Dec;16(12):2257-70. doi: 10.1016/0006-2952(67)90213-4.
3
Biochemical changes in rat liver in response to treatment with drugs and other agents. 3. Effects of centrally acting drugs.大鼠肝脏对药物及其他制剂治疗的生化变化。3. 中枢作用药物的影响。
Biochem Pharmacol. 1969 Feb;18(2):459-73. doi: 10.1016/0006-2952(69)90222-6.
4
Biochemical changes in rat liver in response to treatment with drugs and other agents. I. Effects of anticonvulsant, anti-inflammatory, hypocholesterolaemic and adrenergic beta-blocking agents.大鼠肝脏对药物及其他制剂治疗的生化变化。I. 抗惊厥药、抗炎药、降胆固醇药及肾上腺素能β受体阻滞剂的作用。
Biochem Pharmacol. 1969 Feb;18(2):429-44. doi: 10.1016/0006-2952(69)90220-2.
5
A comparison of mouse and rat liver enzymes and their response to treatment with various compounds.
Biochem Pharmacol. 1972 Apr 1;21(7):1025-9. doi: 10.1016/0006-2952(72)90407-8.
6
Biochemical changes in rat liver in response to treatment with drugs and other agents. IV.大鼠肝脏对药物及其他制剂治疗的生化变化。IV.
Biochem Pharmacol. 1972 Apr 1;21(7):1019-24. doi: 10.1016/0006-2952(72)90406-6.
7
[Action of puromycin, actinomycin D and ethionine on carbohydrate metabolism in the liver of rats fed with thyroid powder].[嘌呤霉素、放线菌素D和乙硫氨酸对喂饲甲状腺粉的大鼠肝脏碳水化合物代谢的作用]
Hoppe Seylers Z Physiol Chem. 1969 Jan;350(1):35-9. doi: 10.1515/bchm2.1969.350.1.35.
8
Comparison of early injury to liver endoplasmic reticulum by halomethanes, hexachloroethane, benzene, toluene, bromobenzene, ethionine, thioacetamide and dimethylnitrosamine.卤代甲烷、六氯乙烷、苯、甲苯、溴苯、乙硫氨酸、硫代乙酰胺和二甲基亚硝胺对肝脏内质网早期损伤的比较。
Biochem Pharmacol. 1972 Oct 1;21(19):2555-61. doi: 10.1016/0006-2952(72)90223-7.
9
[Study on the histopochemistry of oxidative enzymes in thioacetamide induced liver cirrhosis in mice].[硫代乙酰胺诱导小鼠肝硬化氧化酶组织化学研究]
Exp Pathol (Jena). 1970;4(2):101-19.
10
[Enzymatic profile of spleen cells in murine amyloidosis].[小鼠淀粉样变性中脾细胞的酶谱分析]
Biull Eksp Biol Med. 1971 Sep;72(9):114-7.

引用本文的文献

1
Association between Exposure to '-DDT and Its Metabolite '-DDE with Obesity: Integrated Systematic Review and Meta-Analysis.接触γ-滴滴涕及其代谢物γ-滴滴伊与肥胖之间的关联：综合系统评价与荟萃分析。
Environ Health Perspect. 2017 Sep 18;125(9):096002. doi: 10.1289/EHP527.
2
The effect of p,p'-DDT (Dicophane) and its main metabolites on the respiration of rat liver mitochondria.
Arch Toxikol. 1973;31(2):137-45. doi: 10.1007/BF00310391.
3
Liver enzyme induction by 1,1,1-trichloro-2,2-bis-(p-chlorophenyl)ethane (DDT) is accompanied by an increase in the specific activity of elongation factor 1.1,1,1-三氯-2,2-双（对氯苯基）乙烷（滴滴涕）对肝脏酶的诱导作用伴随着延伸因子1比活性的增加。
Biochem J. 1978 Jun 15;172(3):479-86. doi: 10.1042/bj1720479.