通过体外SV40转化的同源胚胎皮肤和皮下组织细胞在仓鼠中诱导各种癌和肉瘤。靶细胞在确定肿瘤形态中的作用。 - Suppr

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超能文献

Induction in hamsters of various carcinomas and sarcomas by in-vitro SV40-transformed homologous embryonic skin and subcutaneous tissue cells. Role of target cells in determining tumor morphology.

作者信息

Diamandopoulos G T, Dalton-Tucker M F

出版信息

Am J Pathol. 1969 Jul;56(1):59-77.

PMID:4307300

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2013576/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/42b1/2013576/aaaf363fdfb4/amjpathol00422-0073-a.jpg

相似文献

Induction in hamsters of various carcinomas and sarcomas by in-vitro SV40-transformed homologous embryonic skin and subcutaneous tissue cells. Role of target cells in determining tumor morphology.通过体外SV40转化的同源胚胎皮肤和皮下组织细胞在仓鼠中诱导各种癌和肉瘤。靶细胞在确定肿瘤形态中的作用。

Am J Pathol. 1969 Jul;56(1):59-77.

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[Formation of tumors from cells transformed in vitro by SV40 virus].[由SV40病毒体外转化的细胞形成肿瘤]

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Effects of chemical carcinogens on neoplastic transformation and morphology of cells in culture.化学致癌物对培养细胞的肿瘤转化及形态的影响。

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[Sensitivity of the embryonic cells of the Djungarian hamster to the mutagenic and transforming action of certain oncogenic viruses in vitro].[黑线仓鼠胚胎细胞在体外对某些致癌病毒诱变和转化作用的敏感性]

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Loss or persistence of the differentiated state of simian virus 40-induced hamster tumor cells before and after serial passage in culture.猿猴病毒40诱导的仓鼠肿瘤细胞在培养中连续传代前后分化状态的丧失或持续存在。

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Histopathology of sarcomas induced in hamsters by clones of in-vitro SV40-transformed homologous heart cells. Attempts to correlate morphologic features and degree ofoncogenic potential.体外SV40转化的同源心脏细胞克隆诱导仓鼠产生的肉瘤的组织病理学。尝试将形态学特征与致癌潜能程度相关联。

Am J Pathol. 1968 Nov;53(5):753-67.

Establishment of a cell line from an inbred mouse strain for viral transformation studies: simian virus 40 transformation and tumor production.从近交系小鼠品系建立用于病毒转化研究的细胞系：猿猴病毒40转化与肿瘤产生

J Natl Cancer Inst. 1968 Dec;41(6):1401-9.

Pathogenesis of malignant neoplasms induced in hamsters by the oncogenic DNA simian virus 40 (SV40).致癌性DNA猿猴病毒40（SV40）诱导仓鼠发生恶性肿瘤的发病机制。

Fed Proc. 1978 May 15;37(7):2082-4.

引用本文的文献

Adenovirus-like transformation of hamster embryo cells mediated by Simian virus 40.由猴病毒40介导的仓鼠胚胎细胞的腺病毒样转化

Am J Pathol. 1973 Apr;71(1):81-92.

本文引用的文献

Propagation in tissue cultures of cytopathogenic agents from patients with measles.麻疹患者细胞病变病原体在组织培养中的传播。

Proc Soc Exp Biol Med. 1954 Jun;86(2):277-86. doi: 10.3181/00379727-86-21073.

Neoplasms in hamsters induced by adenovirus type 12.12型腺病毒诱发的仓鼠肿瘤

Arch Pathol. 1967 Feb;83(2):199-203.

Transformation of rat kidney cells by adenovirus type 12.12型腺病毒对大鼠肾细胞的转化

J Gen Virol. 1968 Mar;2(2):269-72. doi: 10.1099/0022-1317-2-2-269.

A morphologic comparison of tumors produced by type 12 adenovirus and by the HA-12-1T line of adeno-12 tumor cells.12型腺病毒和腺病毒12型肿瘤细胞系HA-12-1T所产生肿瘤的形态学比较。

Cancer Res. 1968 Mar;28(3):577-84.

Plasma cell and lymphocytic reaction in the Syrian hamster to transplanted homologous tumor cells transformed in vitro "spontaneously" or by SV 40.叙利亚仓鼠对体外“自发”或经SV 40转化的同源移植肿瘤细胞的浆细胞和淋巴细胞反应。

Arch Gesamte Virusforsch. 1967;22(1):108-21. doi: 10.1007/BF01240508.

In vitro neoplastic transformation of newborn hamster salivary-gland tissue by oncogenic DNA viruses.致癌DNA病毒对新生仓鼠唾液腺组织的体外肿瘤转化

Cancer. 1966 Oct;19(10):1411-5. doi: 10.1002/1097-0142(196610)19:10<1411::aid-cncr2820191012>3.0.co;2-m.

Histogenesis of malignant neoplasm induced by adenovirus type 12.12型腺病毒诱导的恶性肿瘤的组织发生

Gan. 1966 Feb;57(1):43-52.

Hamster tumor cells doubly transformed by SV40 and polyoma viruses.经SV40病毒和多瘤病毒双重转化的仓鼠肿瘤细胞。

Virology. 1966 Sep;30(1):20-8. doi: 10.1016/s0042-6822(66)81005-x.

Studies on transformation of Syrian hamster cells by simian virus 40 (SV40): acquisition of oncogenicity by virus-exposed cells apparently unassociated with the viral genome.猴病毒40（SV40）对叙利亚仓鼠细胞转化的研究：病毒暴露细胞获得致癌性，这显然与病毒基因组无关。

Proc Natl Acad Sci U S A. 1965 Oct;54(4):1092-9. doi: 10.1073/pnas.54.4.1092.

Antigenic and cultural properties of cells doubly transformed by polyoma virus and SV40.经多瘤病毒和猿猴病毒40双重转化的细胞的抗原性和培养特性

Virology. 1965 Oct;27(2):179-85. doi: 10.1016/0042-6822(65)90157-1.

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