Pathophysiology of hyperuricemia in primary gout.
作者信息
Wyngaarden J B
出版信息
Trans Am Clin Climatol Assoc. 1970;81:161-73.
Abstract
摘要
相似文献
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Pathophysiology of hyperuricemia in primary gout.原发性痛风中高尿酸血症的病理生理学
Trans Am Clin Climatol Assoc. 1970;81:161-73.
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Accelerated erythrocyte 5-phosphoribosyl-1-pyrophosphate synthesis. A familial abnormality associated with excessive uric acid production and gout.
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On the existence of a guanine nucleotide trap, the role of adenosine kinase and a possible cause of excessive purine production in mammalian cells.
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The kinetics of intramolecular distribution of 15N in uric acid following administration of 15N-glycine: preferential labeling of N-(3+9) of uric acid in primary gout and a reappraisal of the "glutamine hypothesis".给予15N-甘氨酸后尿酸中15N的分子内分布动力学：原发性痛风中尿酸N-(3+9)的优先标记及对“谷氨酰胺假说”的重新评估
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Pathophysiology of purine metabolism in man.人类嘌呤代谢的病理生理学
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The kinetics of intramolecular distribution of 15N in uric acid after administration of (15N) glycine. A reappraisal of the significance of preferential labeling of N-(3+9) of uric acid in primary gout.给予（15N）甘氨酸后尿酸中15N的分子内分布动力学。对原发性痛风中尿酸N-（3+9）优先标记意义的重新评估。
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Concentration and synthesis of phosphoribosylpyrophosphate in erythrocytes from normal, hyperuricemic, and gouty subjects.正常、高尿酸血症和痛风患者红细胞中磷酸核糖焦磷酸的浓度与合成
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The Jewish Hospital of St. Louis. Therapeutic grand rounds number 7. An approach to disorders of uric acid metabolism.
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本文引用的文献
1
Purine metabolism in heterozygous carriers of hypoxanthine-guanine phosphoribosyltransferase deficiency.次黄嘌呤 - 鸟嘌呤磷酸核糖转移酶缺乏症杂合子携带者的嘌呤代谢
Science. 1969 Dec 19;166(3912):1533-5. doi: 10.1126/science.166.3912.1533.
2
THE GENETICS OF GOUT AND HYPERURICEMIA-AN ANALYSIS OF NINETEEN FAMILIES.痛风与高尿酸血症的遗传学——对19个家族的分析
J Clin Invest. 1948 Nov;27(6):749-59. doi: 10.1172/JCI102025.
3
The heredity of gout and its relationship to familial hyperuricemia.痛风的遗传及其与家族性高尿酸血症的关系。
Ann Intern Med. 1949 Oct;31(4):595-614. doi: 10.7326/0003-4819-31-4-595.
4
Accelerated turnover of phosphoribosylpyrophosphate, a purine nucleotide precursor, in certain gouty subjects.在某些痛风患者中，嘌呤核苷酸前体磷酸核糖焦磷酸的周转加速。
J Clin Invest. 1962 Sep;41(9):1805-15. doi: 10.1172/JCI104638.
5
6
ACUTE INTERMITTENT PORPHYRIA: THE FIRST "OVERPRODUCTION DISEASE" LOCALIZED TO A SPECIFIC ENZYME.急性间歇性卟啉症：第一种定位到特定酶的“产量过剩疾病”。
Proc Natl Acad Sci U S A. 1965 Apr;53(4):841-7. doi: 10.1073/pnas.53.4.841.
7
8
AN ABNORMALITY OF GLUTAMINE METABOLISM IN PRIMARY GOUT.原发性痛风中谷氨酰胺代谢异常
Am J Med. 1963 Dec;35:820-31. doi: 10.1016/0002-9343(63)90244-4.
9
Uric acid production in gout.痛风中的尿酸生成
J Clin Invest. 1961 Jul;40(7):1304-14. doi: 10.1172/JCI104360.
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