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Parathyroid hormone secretion in familial vitamin-D-resistant rickets.

作者信息

Reitz R E, Weinstein R L

出版信息

N Engl J Med. 1973 Nov 1;289(18):941-5. doi: 10.1056/NEJM197311012891804.

DOI:10.1056/NEJM197311012891804
PMID:4355232
Abstract
摘要

相似文献

1
Parathyroid hormone secretion in familial vitamin-D-resistant rickets.家族性维生素D抵抗性佝偻病中的甲状旁腺激素分泌
N Engl J Med. 1973 Nov 1;289(18):941-5. doi: 10.1056/NEJM197311012891804.
2
Nature of defect responsible for familial vitamin D-resistant rickets (VDRR) based on radioimmunoassay for parathyroid hormone (PTH).基于甲状旁腺激素（PTH）放射免疫测定法的家族性维生素D抵抗性佝偻病（VDRR）相关缺陷的性质
Trans Assoc Am Physicians. 1972;85:172-80.
3
Serum parathyroid hormone concentrations in hypophosphataemic vitamin D resistant rickets.低磷性维生素D抵抗性佝偻病患者的血清甲状旁腺激素浓度
Helv Paediatr Acta. 1974 Aug;29(3):187-94.
4
Serum parathyroid hormone in hypophosphatemic vitamin D-resistant rickets.低磷性维生素D抵抗性佝偻病患者的血清甲状旁腺激素
J Pediatr. 1972 Aug;81(2):294-300. doi: 10.1016/s0022-3476(72)80298-1.
5
[Influence of parathyroid glands on calcium and phosphorus metabolism in hereditary hypophosphatemia].
Pediatr Pol. 1973 Jul;48(7):875-9.
6
[Physiopathological aspects of the parathyroid hormone and calcitonin in childhood].[儿童期甲状旁腺激素和降钙素的生理病理学方面]
Minerva Pediatr. 1979 Dec 15;31(23):1669-76.
7
Exaggerated phosphaturic response to circulating parathyroid hormone in patients with familial X-linked hypophosphatemic rickets.家族性X连锁低磷性佝偻病患者对循环甲状旁腺激素的磷尿反应过度。
J Clin Invest. 1976 Jul;58(1):152-63. doi: 10.1172/JCI108444.
8
Serum 25-hydroxyvitamin D in infantile rickets.婴儿佝偻病中的血清25-羟基维生素D
Pediatrics. 1976 Feb;57(2):221-5.
9
[Vitamin D-resistant rickets].
Tidsskr Nor Laegeforen. 1976 Feb 28;96(6):349-54.
10
Effect of magnesium on phosphorus and calcium metabolism.镁对磷和钙代谢的影响。
Monatsschr Kinderheilkd. 1992 Sep;140(9 Suppl 1):S17-20.

引用本文的文献

1
Primary hyperparathyroidism: four- to eight-year postoperative follow-up demonstrating persistent functional insignificance of microscopic parathyroid hyperplasia and decreased autonomy of parathyroid hormone release.原发性甲状旁腺功能亢进症：术后4至8年的随访表明，微小甲状旁腺增生持续无功能意义，且甲状旁腺激素释放自主性降低。
Ann Surg. 1981 Oct;194(4):429-37. doi: 10.1097/00000658-198110000-00006.
2
Abnormal parathyroid function in the X-linked hypophosphatemic mouse.
Calcif Tissue Int. 1985 Jul;37(4):418-22. doi: 10.1007/BF02553712.
3
Regulation of circulating parathyroid hormone levels: normal physiology and consequences in disorders of mineral metabolism.循环甲状旁腺激素水平的调节：矿物质代谢紊乱中的正常生理及影响
Klin Wochenschr. 1975 Oct 15;53(20):939-54. doi: 10.1007/BF01614437.
4
Phosphorus deficiency, parathyroid hormone and bone resorption in the growing rat.生长中大鼠的磷缺乏、甲状旁腺激素与骨吸收
Calcif Tissue Res. 1976 Jun 14;20(3):235-49. doi: 10.1007/BF02546412.
5
Evidence for an intrinsic renal tubular defect in mice with genetic hypophosphatemic rickets.遗传性低磷性佝偻病小鼠肾小管内在缺陷的证据。
J Clin Invest. 1979 Jun;63(6):1203-10. doi: 10.1172/JCI109415.
6
Micropuncture study of renal phosphorus transport in hypophosphatemic vitamin D resistant rickets mice.低磷血症性维生素D抵抗性佝偻病小鼠肾脏磷转运的微穿刺研究
Pflugers Arch. 1977 Oct 19;371(1-2):33-8. doi: 10.1007/BF00580769.
7
Recessive hypophosphataemic rickets, and possible aetiology of the 'vitamin D-resistant' syndrome.隐性低磷血症性佝偻病以及“维生素D抵抗”综合征的可能病因。
Arch Dis Child. 1976 May;51(5):360-5. doi: 10.1136/adc.51.5.360.
8
Exaggerated phosphaturic response to circulating parathyroid hormone in patients with familial X-linked hypophosphatemic rickets.家族性X连锁低磷性佝偻病患者对循环甲状旁腺激素的磷尿反应过度。
J Clin Invest. 1976 Jul;58(1):152-63. doi: 10.1172/JCI108444.