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[症状性面部半侧萎缩的发病机制]

[Pathogenesis of symptomatic facial hemiatrophy].

作者信息

Tebloev I K, Kalashnikov Iu D

出版信息

Zh Nevropatol Psikhiatr Im S S Korsakova. 1979;79(4):413-6.

PMID:442909
Abstract

The data on clinical and electroencephalographic examination of 19 patients are presented. The patients developed facial hemiatrophy after they had sustained ganglionitis of the superior cervical sympathetic ganglion, stem encephalitis, trifacial neuralgia (neuritic stage), tumours of the Gasserian ganglion and syringobulbia. A pathogenetic relationship between the development of symptomatic facial hemiatrophy and lesions of the vegetative and somatic apparatus in the cerebral (mesencephalotruncal area) and extracerebral (superior cervical sympathetic ganglion) structures of the nervous system is noted.

摘要

本文呈现了19例患者的临床及脑电图检查数据。这些患者在发生颈上交感神经节神经炎、脑干脑炎、三叉神经痛(神经炎期)、半月神经节肿瘤及延髓空洞症后出现了面部偏侧萎缩。文中指出,症状性面部偏侧萎缩的发生与神经系统脑内(中脑延髓区)和脑外(颈上交感神经节)结构中的自主和躯体器官病变之间存在病因学关联。

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