Swash M
J Neurol Neurosurg Psychiatry. 1974 Jan;37(1):21-6. doi: 10.1136/jnnp.37.1.21.
A patient with hydrocephalus due to aqueductal occlusion is described in whom the Sylvian aqueduct syndrome appeared during a sudden increase in intracranial pressure. The ocular signs resolved completely when the hydrocephalus was relieved. Marked dilatation of the posterior part of the third ventricle and of the rostral aqueduct with axial displacement of these structures was demonstrated radiologically. It is suggested that the ocular signs in this case were the result of periaqueductal dysfunction due to assimilation and dilatation of the aqueduct, with secondary tentorial block. This abnormality may be the cause of the similar abnormalities commonly found in noncommunicating hydrocephalus in both infants and adults.
本文描述了一名因导水管阻塞导致脑积水的患者，其在颅内压突然升高时出现了西尔维安导水管综合征。当脑积水缓解时，眼部体征完全消失。影像学检查显示第三脑室后部和导水管前部明显扩张，这些结构发生轴向移位。提示该病例的眼部体征是由于导水管同化和扩张导致导水管周围功能障碍，并继发小脑幕阻塞所致。这种异常可能是婴儿和成人非交通性脑积水常见类似异常的原因。
