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孤立性促性腺激素缺乏男孩的生长模式。

Growth pattern of boys with isolated gonadotropin deficiency.

作者信息

Kaushanski A, Laron Z

出版信息

Isr J Med Sci. 1979 Jun;15(6):518-21.

PMID:457385
Abstract

The linear growth and skeletal maturation of 19 boys with isolated gonadotropin deficiency, including four with anosmia (Kallmann's syndrome), are presented. With the exception of one prepubertal-age boy (less than or equal to 10 years) and five pubertal-age boys (greater than 10 years), all heights were within normal limits, but below the 50th percentile. With one exception, the bone age was retarded in all boys, even in prepuberty.

摘要

本文报告了19例孤立性促性腺激素缺乏症男孩的线性生长和骨骼成熟情况,其中包括4例嗅觉缺失(卡尔曼综合征)的男孩。除1例青春期前男孩(小于或等于10岁)和5例青春期男孩(大于10岁)外,所有男孩的身高均在正常范围内,但低于第50百分位数。除1例外,所有男孩的骨龄均延迟,即使在青春期前也是如此。

相似文献

1
Growth pattern of boys with isolated gonadotropin deficiency.孤立性促性腺激素缺乏男孩的生长模式。
Isr J Med Sci. 1979 Jun;15(6):518-21.
2
Acromegalic features in growth hormone (GH)-deficient patients after long-term GH therapy.长期生长激素(GH)治疗后生长激素缺乏患者出现肢端肥大症特征。
Clin Endocrinol (Oxf). 2003 Dec;59(6):788-92. doi: 10.1046/j.1365-2265.2003.01899.x.
3
Kallmann's syndrome: reproductive success.卡尔曼综合征:生殖成功。
N J Med. 1990 Aug;87(8):637-9.
4
Normal sexual maturation.正常性成熟。
Pediatrician. 1987;14(4):191-201.
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Presence of magnetic resonance imaging abnormalities of the hypothalamic-pituitary axis is a significant determinant of the first 3 years growth response to human growth hormone treatment in prepubertal children with nonacquired growth hormone deficiency.下丘脑 - 垂体轴磁共振成像异常的存在是青春期前非获得性生长激素缺乏儿童对人生长激素治疗最初3年生长反应的重要决定因素。
Clin Endocrinol (Oxf). 2003 May;58(5):647-52.
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Correlations between skeletal muscle mass and bone mass in children 6-18 years: influences of sex, ethnicity, and pubertal status.6至18岁儿童骨骼肌质量与骨质量的相关性:性别、种族和青春期状态的影响。
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Hum Reprod Update. 2004 Mar-Apr;10(2):135-47. doi: 10.1093/humupd/dmh012.
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Effect of long-term growth hormone therapy on bone age and pubertal maturation in boys with and without classic growth hormone deficiency.长期生长激素治疗对伴有和不伴有典型生长激素缺乏症男孩骨龄和青春期成熟的影响。
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引用本文的文献

1
Childhood growth in boys with congenital hypogonadotropic hypogonadism.先天性低促性腺激素性性腺功能减退男孩的儿童期生长情况
Pediatr Res. 2016 May;79(5):705-9. doi: 10.1038/pr.2015.278. Epub 2015 Dec 31.
2
Recent experimental and clinical findings in the skeleton associated with loss of estrogen hormone or estrogen receptor activity.与雌激素激素或雌激素受体活性丧失相关的骨骼的最近实验和临床发现。
J Steroid Biochem Mol Biol. 2010 Feb 28;118(4-5):264-72. doi: 10.1016/j.jsbmb.2009.10.016. Epub 2009 Nov 10.
3
Case report 848. Kallman's syndrome: hypogonadotropic hypogonadism with delayed closure of epiphyseal growth zones, resulting in epiphysiolysis of the left proximal femoral epiphysis after trauma.
病例报告848. 卡尔曼综合征:促性腺激素缺乏性性腺功能减退伴骨骺生长区闭合延迟,外伤后导致左股骨近端骨骺分离。
Skeletal Radiol. 1994 Jul;23(5):385-7. doi: 10.1007/BF02416999.
4
Adrenarche and skeletal maturation during luteinizing hormone releasing hormone analogue suppression of gonadarche.促性腺激素释放激素类似物抑制青春期时的肾上腺功能初现和骨骼成熟。
J Clin Invest. 1986 Jan;77(1):121-6. doi: 10.1172/JCI112265.
5
Growth in patients with isolated gonadotrophin deficiency.孤立性促性腺激素缺乏患者的生长情况。
Arch Dis Child. 1992 Apr;67(4):513-6. doi: 10.1136/adc.67.4.513.