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[Effect of benziodaron on purine metabolism in gout patients and healthy subjects].

作者信息

Müller M M, Bresnik W

出版信息

Klin Wochenschr. 1972 Nov 1;50(21):1015-6. doi: 10.1007/BF01486997.

DOI:10.1007/BF01486997
PMID:4637344
Abstract
摘要

相似文献

1
[Effect of benziodaron on purine metabolism in gout patients and healthy subjects].[苯碘达隆对痛风患者和健康受试者嘌呤代谢的影响]
Klin Wochenschr. 1972 Nov 1;50(21):1015-6. doi: 10.1007/BF01486997.
2
[Clinical biochemistry of gout].
Dtsch Med J. 1969 May 27;20(10):336-41.
3
The Jewish Hospital of St. Louis. Therapeutic grand rounds number 7. An approach to disorders of uric acid metabolism.
Arch Intern Med. 1974 Sep;134(3):579-85.
4
Influence of a uricosuric drug on connective tissue metabolism.一种促尿酸排泄药物对结缔组织代谢的影响。
Adv Exp Med Biol. 1974;41:693-8. doi: 10.1007/978-1-4757-1433-3_42.
5
Purine base incorporation into erythrocyte nucleotides and erythrocyte phosphoribosyltransferase activity in primary gout.原发性痛风中嘌呤碱基掺入红细胞核苷酸及红细胞磷酸核糖基转移酶活性
Rev Eur Etud Clin Biol. 1971 Feb;16(2):147-51.
6
Azaguanine-resistance as a manifestation of a new form of metabolic overproduction of uric acid.
Am J Med. 1972 Apr;52(4):547-55. doi: 10.1016/0002-9343(72)90046-0.
7
Chemiluminescent assays in the study of purine metabolism.嘌呤代谢研究中的化学发光分析
Adv Exp Med Biol. 1989;253A:411-5. doi: 10.1007/978-1-4684-5673-8_67.
8
Gout with purine overproduction due to increased phosphoribosylpyrophosphate synthetase activity.由于磷酸核糖焦磷酸合成酶活性增加导致嘌呤产生过多的痛风。
Am J Med. 1973 Aug;55(2):232-42. doi: 10.1016/0002-9343(73)90174-5.
9
Increased PP-ribose-P synthetase activity: a genetic abnormality leading to excessive purine production and gout.磷酸核糖焦磷酸合成酶活性增加：一种导致嘌呤过度生成和痛风的基因异常。
Adv Exp Med Biol. 1973;41:307-15. doi: 10.1007/978-1-4684-3294-7_37.
10
Glycine-C-14 incorporation into nucleic acid purine by leucocytes obtained from normal and gouty subjects.甘氨酸-C-14被正常人和痛风患者的白细胞掺入核酸嘌呤中。
Ann Rheum Dis. 1969 May;28(3):275-80. doi: 10.1136/ard.28.3.275.

引用本文的文献

1
[Action of benzbromarone on the renal excretion of allantoin in normouricemic and hyperuricemic humans (author's transl)].
Klin Wochenschr. 1977 Nov 1;55(21):1071-3. doi: 10.1007/BF01489483.
2
[Purine nucleotide synthesis in human liver an its influence by benzbromarone (author's transl)].
Klin Wochenschr. 1978 Aug 15;56(16):835-7. doi: 10.1007/BF01489718.

本文引用的文献

1
Standardization of hemoglobinometry. II. The hemiglobincyanide method.血红蛋白测定法的标准化。II. 高铁氰化血红蛋白法。
Clin Chim Acta. 1961 Jul;6:538-44. doi: 10.1016/0009-8981(61)90145-0.
2
Enzymatic synthesis of pyrimidine nucleotides; orotidine-5'-phosphate and uridine-5'-phosphate.嘧啶核苷酸的酶促合成；乳清苷-5'-磷酸和尿苷-5'-磷酸。
J Biol Chem. 1955 Jul;215(1):403-51.
3
The regulation of activity of phosphoribosyl-pyrophosphate amidotransferase by purine ribonacleotides: a potential feedback control of purine biosyntoesis.
嘌呤核糖核苷酸对磷酸核糖焦磷酸酰胺转移酶活性的调节：嘌呤生物合成的一种潜在反馈控制。
J Biol Chem. 1959 Jun;234(6):1492-6.
4
Enzymatic determination of uric acid; with detailed directions.尿酸的酶法测定；详细说明。
Scand J Clin Lab Invest. 1953;5(3):273-80. doi: 10.3109/00365515309094197.
5
Inherited disorder of purine metabolism. Correlation between central nervous system dysfunction and biochemical defects.嘌呤代谢的遗传性疾病。中枢神经系统功能障碍与生化缺陷之间的关联。
JAMA. 1967 Oct 16;202(3):175-7. doi: 10.1001/jama.202.3.175.
6
[Effect of benzbromaronum on the renal uric acid excretion in healthy persons].
Klin Wochenschr. 1970 Apr 1;48(7):426-32. doi: 10.1007/BF01487010.
7
[Clinical and biological effects of a new uric acid lowering substance (benziodaronum) in gout. Results of a 2-year-long study].
Verh Dtsch Ges Rheumatol. 1969;1:361-70.
8
[Clinical biochemistry of gout].
Dtsch Med J. 1969 May 27;20(10):336-41.
9
Purification and properties of 5-phosphoribosyl pyrophosphate amidotransferase from adenocarcinoma 755 cells.
Biochemistry. 1969 Jan;8(1):122-30. doi: 10.1021/bi00829a017.
10
A specific enzyme defect in gout associated with overproduction of uric acid.痛风中与尿酸生成过多相关的一种特定酶缺陷。
Proc Natl Acad Sci U S A. 1967 Jun;57(6):1735-9. doi: 10.1073/pnas.57.6.1735.