Bersenev V A
Zh Nevropatol Psikhiatr Im S S Korsakova. 1979;79(7):884-8.
On the basis of literature analysis, clinical observations, and examinations of the spinal cord ganglia of man (autopsy material) and experimental animals (dogs) performed with the use of a set of naurohistological and cytochemical methods an original explanation of the pathogenesis of the Head zones is suggested. The principal link in the formation of the viscero-cutaneous and viscero-sclerotomic sensation syndrome are transitory presynaptic terminals formed by the axons of the vegetative sensor neurons (cells of Dogel type 2) on the surface of the somatic receptor neurons innervating the skin and the sclerotomic tissues, and responding to temperature and pain. Within the zone of the innervation by the dendrites of these somatic receptor neurons (cutaneous and sclerotomic) a reflected viscero-sensory syndrome is formed. The formation of these viscero-cutaneous and viscero-sclerotomic syndromes proceeds in 4 steps.
基于文献分析、临床观察以及运用一系列神经组织学和细胞化学方法对人类（尸检材料）和实验动物（狗）的脊髓神经节进行的检查，本文提出了对海德带发病机制的一种原创性解释。内脏 - 皮肤和内脏 - 硬节感觉综合征形成的主要环节是由植物性感觉神经元（多吉尔2型细胞）的轴突在支配皮肤和硬节组织的躯体受体神经元表面形成的短暂性突触前终末，这些终末对温度和疼痛有反应。在这些躯体受体神经元（皮肤和硬节）的树突支配区域内，形成了一种反射性内脏感觉综合征。这些内脏 - 皮肤和内脏 - 硬节综合征的形成过程分4个步骤。
