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Morphogenesis of the malformations induced in rat embryos by maternal hypervitaminosis A.

作者信息

Morriss G M

出版信息

J Anat. 1972 Nov;113(Pt 2):241-50.

PMID:4664235
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1271684/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/9e49e70d049c/janat00408-0091-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/adbb169f29cd/janat00408-0085-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/02c7b4442d28/janat00408-0085-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/03a748a4cb08/janat00408-0085-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/6e6ad4018e89/janat00408-0085-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/cf9c2ac2a61f/janat00408-0085-e.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0dc5407897d5/janat00408-0087-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0411d93495ab/janat00408-0087-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/a435e38d5989/janat00408-0087-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/43eb9c617820/janat00408-0087-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/ad75e33df9f6/janat00408-0089-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0d0e5de5ef64/janat00408-0089-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/a1a388c396b0/janat00408-0089-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0dd342088031/janat00408-0091-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/042d052ef425/janat00408-0091-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/9e49e70d049c/janat00408-0091-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/adbb169f29cd/janat00408-0085-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/02c7b4442d28/janat00408-0085-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/03a748a4cb08/janat00408-0085-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/6e6ad4018e89/janat00408-0085-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/cf9c2ac2a61f/janat00408-0085-e.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0dc5407897d5/janat00408-0087-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0411d93495ab/janat00408-0087-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/a435e38d5989/janat00408-0087-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/43eb9c617820/janat00408-0087-d.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/ad75e33df9f6/janat00408-0089-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0d0e5de5ef64/janat00408-0089-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/a1a388c396b0/janat00408-0089-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/0dd342088031/janat00408-0091-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/042d052ef425/janat00408-0091-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/94f2/1271684/9e49e70d049c/janat00408-0091-c.jpg

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本文引用的文献

1
[Cleft palate in the rat embryo caused by maternal hypervitaminosis A].[母体维生素A过多症导致大鼠胚胎腭裂]
C R Seances Soc Biol Fil. 1954 Nov;148(21-22):1742-3.
2
THE MECHANISM OF VITAMIN A INDUCED TERATOGENESIS.维生素A诱导致畸的机制。
J Embryol Exp Morphol. 1964 Jun;12:263-71.
3
Observations on the effect of experimental endocrine procedures on the teratogenic action of hypervitaminosis A in the rat.关于实验性内分泌操作对大鼠维生素A过多症致畸作用影响的观察
J Anat. 2016 Jul;229(1):2-31. doi: 10.1111/joa.12489.
4
Retinoic acid deficiency impairs the vestibular function.视黄酸缺乏会损害前庭功能。
J Neurosci. 2013 Mar 27;33(13):5856-66. doi: 10.1523/JNEUROSCI.4618-12.2013.
5
Does the cranial mesenchyme contribute to neural fold elevation during neurulation?颅间充质在神经胚形成过程中对神经褶抬高有贡献吗？
Birth Defects Res A Clin Mol Teratol. 2012 Oct;94(10):841-8. doi: 10.1002/bdra.23073. Epub 2012 Sep 3.
6
Retinoic acid downregulates microRNAs to induce abnormal development of spinal cord in spina bifida rat model.维甲酸下调微小RNA以诱导脊柱裂大鼠模型脊髓异常发育。
Childs Nerv Syst. 2008 Apr;24(4):485-92. doi: 10.1007/s00381-007-0520-5. Epub 2007 Oct 26.
7
Retinoic acid repression of bone morphogenetic protein 4 in inner ear development.维甲酸对内耳发育过程中骨形态发生蛋白4的抑制作用。
Mol Cell Biol. 2003 Apr;23(7):2277-86. doi: 10.1128/MCB.23.7.2277-2286.2003.
8
Expression of Fgf-3 in relation to hindbrain segmentation, otic pit position and pharyngeal arch morphology in normal and retinoic acid-exposed mouse embryos.正常及视黄酸处理的小鼠胚胎中Fgf-3的表达与后脑分节、耳窝位置及咽弓形态的关系
Anat Embryol (Berl). 1996 Jul;194(1):13-22. doi: 10.1007/BF00196311.
9
Notochordal alterations in axial skeletal-neural dysraphic disorders.
Virchows Arch A Pathol Anat Histopathol. 1993;422(2):97-8. doi: 10.1007/BF01607160.
10
Role of the notochord in the development of cephalic structures in normal and anencephalic human fetuses.脊索在正常及无脑儿胎儿头部结构发育中的作用。
Virchows Arch A Pathol Anat Histopathol. 1993;422(2):161-8. doi: 10.1007/BF01607168.
Biol Neonat. 1961 Dec;3:330-42. doi: 10.1159/000239854.
4
[Teratogenic action of hypervitaminosis A in the mouse as a function of th embryonic stage].[小鼠维生素A过多症的致畸作用与胚胎发育阶段的关系]
C R Seances Soc Biol Fil. 1960;154:1353-5.
5
Effect of vitamin B complex on the teratogenic activity of hypervitaminosis A.复合维生素B对维生素A过多症致畸活性的影响。
Nature. 1958 Oct 4;182(4640):940. doi: 10.1038/182940a0.
6
Influence of 4-methyl-2-thiouracil on the teratogenic activity of hypervitaminosis-A.4-甲基-2-硫脲嘧啶对维生素A过多症致畸活性的影响。
Nature. 1958 Apr 5;181(4614):992-3. doi: 10.1038/181992a0.
7
Influence of cortisone on teratogenic effects of hypervitaminosis-A.可的松对维生素A过多症致畸作用的影响。
Br Med J. 1957 Jul 27;2(5038):196-7. doi: 10.1136/bmj.2.5038.196.
8
[Hypervitaminosis A and abnormalities in rat fetuses].[维生素A过多症与大鼠胎儿异常]
Int Z Vitaminforsch Beih. 1955;26(1-2):10-8.
9
Excessive intake of vitamin A as a cause of congenital anomalies in the rat.大鼠先天性异常的一个病因：维生素A摄入过量。
Science. 1953 May 15;117(3046):535-6. doi: 10.1126/science.117.3046.535.
10
A radioautographic study of the migration and fate of cranial neural crest cells in the chick embryo.鸡胚中颅神经嵴细胞迁移和归宿的放射自显影研究。
Anat Rec. 1966 Oct;156(2):143-55. doi: 10.1002/ar.1091560204.