Mechanisms and consequences of the induction of microsomal enzymes of mammalian liver.
作者信息
Parke D V
出版信息
Biochem J. 1972 Nov;130(2):53P-55P. doi: 10.1042/bj1300053p.
Abstract
摘要
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本文引用的文献
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Studies on the induction of CO-binding pigments in liver microsomes by phenobarbital and 3-methylcholanthrene.苯巴比妥和3-甲基胆蒽对肝微粒体中一氧化碳结合色素诱导作用的研究。
Biochem Biophys Res Commun. 1967 Nov 30;29(4):521-6. doi: 10.1016/0006-291x(67)90515-3.
2
DIFFERENCES IN HEPATIC DRUG METABOLISM IN VARIOUS RABBIT STRAINS BEFORE AND AFTER PRETREATMENT WITH PHENOBARBITAL.苯巴比妥预处理前后不同兔种肝脏药物代谢的差异。
Proc Soc Exp Biol Med. 1965 Apr;118:872-5. doi: 10.3181/00379727-118-29994.
3
PUROMYCIN INHIBITION OF ENZYME INDUCTION BY 3-METHYLCHOLANTHRENE AND PHENOBARBITAL.嘌呤霉素对3-甲基胆蒽和苯巴比妥诱导酶的抑制作用。
J Biol Chem. 1963 Nov;238:3682-5.
4
The effect of phenobarbital on the synthesis of microsomal phospholipid in female and male rats.苯巴比妥对雌性和雄性大鼠微粒体磷脂合成的影响。
Biochem Biophys Res Commun. 1966 Sep 8;24(5):639-43. doi: 10.1016/0006-291x(66)90371-8.
5
Microsome-specific stimulation by phenobarbital of amino acid incorporation in vivo.苯巴比妥对体内氨基酸掺入的微粒体特异性刺激作用。
Biochem Pharmacol. 1965 Jul;14(7):1164-6. doi: 10.1016/0006-2952(65)90047-x.
6
7
Activation of genetic transcription in rat liver chromatin by 3-methylcholanthrene.3-甲基胆蒽对大鼠肝脏染色质中基因转录的激活作用。
Mol Pharmacol. 1969 May;5(3):219-26.
8
Effect of sodium phenobarbital on bilirubin metabolism in an infant with congenital, nonhemolytic, unconjugated hyperbilirubinemia, and kernicterus.苯巴比妥钠对一名患有先天性、非溶血性、非结合性高胆红素血症及核黄疸婴儿胆红素代谢的影响。
J Clin Invest. 1969 Jan;48(1):42-55. doi: 10.1172/JCI105973.
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Differences in the kinetics of benzpyrene hydroxylation by hepatic drug-metabolizing enzymes from phenobarbital and 3-methylcholanthrene-treated rats.苯巴比妥和3-甲基胆蒽处理的大鼠肝脏药物代谢酶对苯并芘羟基化动力学的差异。
Biochem Biophys Res Commun. 1968 Mar 12;30(5):588-93. doi: 10.1016/0006-291x(68)90094-6.
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Induction and repression of microsomal drug-metabolizing enzymes by polycyclic hydrocarbons and phenobarbital: theoretical models.
J Theor Biol. 1971 Dec;33(3):517-37. doi: 10.1016/0022-5193(71)90094-4.
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