长春新碱诱导艾氏腹水癌细胞中甲氨蝶呤摄取增加的特征。 - Suppr

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超能文献

Characteristics of the vincristine-induced augmentation of methotrexate uptake in Ehrlich ascites tumor cells.

作者信息

Fyfe M J, Goldman I D

出版信息

J Biol Chem. 1973 Jul 25;248(14):5067-73.

PMID:4736889

Abstract

摘要

相似文献

Characteristics of the vincristine-induced augmentation of methotrexate uptake in Ehrlich ascites tumor cells.长春新碱诱导艾氏腹水癌细胞中甲氨蝶呤摄取增加的特征。

J Biol Chem. 1973 Jul 25;248(14):5067-73.

The characteristics of the membrane transport of amethopterin and the naturally occurring folates.氨甲蝶呤和天然存在的叶酸的膜转运特性。

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Teniposide (VM-26)- and etoposide (VP-16-213)-induced augmentation of methotrexate transport and polyglutamylation in Ehrlich ascites tumor cells in vitro.替尼泊苷（VM - 26）和依托泊苷（VP - 16 - 213）在体外诱导艾氏腹水癌细胞中氨甲蝶呤转运及多聚谷氨酰化作用增强。

Cancer Res. 1982 Sep;42(9):3648-53.

A model system for the study of heteroexchange diffusion: methotrexate-folate interactions in L1210 leukemia and Ehrlich ascites tumor cells.用于研究异质交换扩散的模型系统：甲氨蝶呤与叶酸在L1210白血病细胞和艾氏腹水瘤细胞中的相互作用

Biochim Biophys Acta. 1971 Jun 1;233(3):624-34. doi: 10.1016/0005-2736(71)90162-3.

The mechanism of action of methotrexate. II. Augmentation by vincristine of inhibition of deoxyribonucleic aic synthesis by methotrexate in Ehrlich ascites tumor cells.甲氨蝶呤的作用机制。II. 长春新碱增强甲氨蝶呤对艾氏腹水瘤细胞脱氧核糖核酸合成的抑制作用。

Mol Pharmacol. 1974 Mar;10(2):275-82.

Amethopterin transport in Ehrlich ascites carcinoma and L1210 cells.氨甲蝶呤在艾氏腹水癌和L1210细胞中的转运。

Cancer Res. 1967 Sep;27(9):1539-43.

Transport energetics of the folic acid analogue, methotrexate, in L1210 leukemia cells. Enhanced accumulation by metabolic inhibitors.叶酸类似物甲氨蝶呤在L1210白血病细胞中的转运能量学。代谢抑制剂增强其积累。

J Biol Chem. 1969 Jul 25;244(14):3779-85.

Different effects of vincristine on methotrexate uptake by L1210 cells and mouse intestinal epithelia in vitro and in vivo.长春新碱对L1210细胞和小鼠肠上皮细胞在体外和体内摄取甲氨蝶呤的不同影响。

Cancer Res. 1979 Jun;39(6 Pt 1):2106-12.

Intracellular phosphate and its possible role as an exchange anion for active transport of methotrexate in L1210 cells.

Biochem Biophys Res Commun. 1982 Jan 29;104(2):474-82. doi: 10.1016/0006-291x(82)90661-1.

Serum protein reduction of the enhancement of methotrexate accumulation by vincristine and 4'-demethylepipodophyllotoxin in the Ehrlich ascites tumor cell in vitro.长春新碱和4'-去甲基表鬼臼毒素增强甲氨蝶呤在艾氏腹水瘤细胞体外蓄积作用时的血清蛋白减少情况。

Cancer Res. 1985 Dec;45(12 Pt 1):6290-2.

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Coadministration of vindesine with high-dose methotrexate therapy increases acute kidney injury via BCRP, MRP2, and OAT1/OAT3.长春碱与大剂量甲氨蝶呤联合治疗通过 BCRP、MRP2 和 OAT1/OAT3 增加急性肾损伤。

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Interactions of vinblastine and vincristine with methotrexate transport in isolated rat hepatocytes.长春碱和长春新碱与甲氨蝶呤在分离的大鼠肝细胞中转运的相互作用。

Cancer Chemother Pharmacol. 1993;32(3):209-14. doi: 10.1007/BF00685837.

Methotrexate-vindesine association in the treatment of head and neck cancer influence of vindesine on methotrexate's pharmacokinetic behavior.

Cancer Chemother Pharmacol. 1984;12(2):120-4. doi: 10.1007/BF00254603.

Study of fluxes at low concentrations of L-tri-iodothyronine with rat liver cells and their plasma-membrane vesicles. Evidence for the accumulation of the hormone against a gradient.低浓度L-三碘甲状腺原氨酸与大鼠肝细胞及其质膜囊泡的通量研究。激素逆浓度梯度积累的证据。

Biochem J. 1981 Sep 15;198(3):457-66. doi: 10.1042/bj1980457.

Lysosomal physiology in Tetrahymena. 3. Pharmacological studies on acid hydrolase release and the ingestion and egestion of dimethylbenzanthracene particles.四膜虫的溶酶体生理学。3. 关于酸性水解酶释放以及二甲基苯并蒽颗粒摄取与排出的药理学研究。

J Cell Biol. 1974 Sep;62(3):844-59. doi: 10.1083/jcb.62.3.844.

Characterization of the multiple transport routes for methotrexate in L1210 cells using phthalate as a model anion substrate.

J Membr Biol. 1985;85(3):263-8. doi: 10.1007/BF01871521.

Methotrexate-vindesine association in leukemia: pharmacokinetic study.甲氨蝶呤与长春地辛联合治疗白血病的药代动力学研究。

Med Oncol Tumor Pharmacother. 1985;2(2):99-102. doi: 10.1007/BF02934855.

Methotrexate-vindesine association in head and neck cancer: modification of methotrexate's hydroxylation in presence of vindesine.

Cancer Chemother Pharmacol. 1986;17(2):171-6. doi: 10.1007/BF00306749.

Flow cytometric analysis of marrow cell kinetics in children treated with high-dose MTX and CF rescue.

Cancer Chemother Pharmacol. 1986;16(3):277-81. doi: 10.1007/BF00293992.

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