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The effect of thiamine treatment on the activity of pyruvate dehydrogenase: relation of the treatment of Leigh's encephalomyelopathy.

作者信息

Hommes F A, Berger R, Luit-de-Haan G

出版信息

Pediatr Res. 1973 Jul;7(7):616-9. doi: 10.1203/00006450-197307000-00003.

DOI:10.1203/00006450-197307000-00003
PMID:4736981
Abstract
摘要

相似文献

1
The effect of thiamine treatment on the activity of pyruvate dehydrogenase: relation of the treatment of Leigh's encephalomyelopathy.
Pediatr Res. 1973 Jul;7(7):616-9. doi: 10.1203/00006450-197307000-00003.
2
Biochemical studies and therapy in subacute necrotizing encephalomyelopathy (Leigh's syndrome).亚急性坏死性脑脊髓病( Leigh 综合征)的生化研究与治疗
J Pediatr. 1972 Jul;81(1):39-44. doi: 10.1016/s0022-3476(72)80371-8.
3
Further speculation on the pathogenesis of Leigh's encephalomyelopathy.
J Pediatr. 1973 Mar;82(3):541-2. doi: 10.1016/s0022-3476(73)80144-1.
4
[Leigh's syndrome].
Monatsschr Kinderheilkd (1902). 1974 Jan;122(1):44-5.
5
Efficacy of recommended therapeutic regimens in Leigh's disease.推荐治疗方案对Leigh病的疗效。
Dev Med Child Neurol. 1974 Jun;16(3):362-4. doi: 10.1111/j.1469-8749.1974.tb03348.x.
6
Subacute necrotizing encephalomyelopathy (Leigh's disease): a consideration of clinical features and etiology.亚急性坏死性脑脊髓病( Leigh 病):临床特征与病因学探讨
Dev Med Child Neurol. 1972 Feb;14(1):87-101. doi: 10.1111/j.1469-8749.1972.tb02563.x.
7
Pathogenesis of Leigh's encephalomyelopathy.Leigh 脑脊髓病的发病机制。
J Pediatr. 1972 Jul;81(1):189-90. doi: 10.1016/s0022-3476(72)80428-1.
8
[Subacute necrotizing encephalomyelopathy (Leigh's disease) (author's transl)].
Monatsschr Kinderheilkd (1902). 1975 Feb;123(2):84-90.
9
[Subacute necrotizing encephalomyelopathy in identical twins. Pathogenetic and therapeutic aspects].[同卵双胞胎的亚急性坏死性脑脊髓病。发病机制及治疗方面]
Monatsschr Kinderheilkd (1902). 1973 Jul;121(7):294-6.
10
Subacute necrotizing encephalomyelopathy. Clinical, ultrastructural, biochemical and therapeutic studies in an infant.亚急性坏死性脑脊髓病。一名婴儿的临床、超微结构、生化及治疗研究。
Acta Paediatr Scand. 1975 Sep;64(5):755-62. doi: 10.1111/j.1651-2227.1975.tb03916.x.

引用本文的文献

1
Organic cation transporter 1 (OCT1) modulates multiple cardiometabolic traits through effects on hepatic thiamine content.有机阳离子转运蛋白 1(OCT1)通过影响肝脏硫胺素含量来调节多种心脏代谢特征。
PLoS Biol. 2018 Apr 16;16(4):e2002907. doi: 10.1371/journal.pbio.2002907. eCollection 2018 Apr.
2
Hyperalaninemia hyperpyruvicemia and lactic acidosis due to pyruvate carboxylase deficiency of the liver; treatment with thiamine and lipoic acid.由于肝脏丙酮酸羧化酶缺乏导致的高丙氨酸血症、高丙酮酸血症和乳酸酸中毒;硫胺素和硫辛酸治疗。
Eur J Pediatr. 1976 May 4;122(2):159-68. doi: 10.1007/BF00466274.
3
Thiamine dependency in a patient with congenital lacticacidaemia due to pyruvate dehydrogenase deficiency.
一名因丙酮酸脱氢酶缺乏导致先天性乳酸性酸中毒患者的硫胺素依赖性。
Agents Actions. 1977 Sep;7(3):405-10. doi: 10.1007/BF01969575.