• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Induced alterations in the rate-limiting enzymes of hepatic cholesterol and bile acid synthesis in the hamster.

作者信息

Schoenfield L J, Bonorris G G, Ganz P

出版信息

J Lab Clin Med. 1973 Dec;82(6):858-68.

PMID:4758737
Abstract
摘要

相似文献

1
Induced alterations in the rate-limiting enzymes of hepatic cholesterol and bile acid synthesis in the hamster.仓鼠肝脏胆固醇和胆汁酸合成限速酶的诱导性改变。
J Lab Clin Med. 1973 Dec;82(6):858-68.
2
Effects of treatment with deoxycholic acid and chenodeoxycholic acid on the hepatic synthesis of cholesterol and bile acids in healthy subjects.脱氧胆酸和鹅去氧胆酸治疗对健康受试者肝脏胆固醇和胆汁酸合成的影响。
Hepatology. 2001 May;33(5):1189-93. doi: 10.1053/jhep.2001.23790.
3
Interaction of pituitary gland and bile acid pool size on the circadian rhythm of cholesterol-7alpha-hydroxylase activity of rats.
Int J Chronobiol. 1974;2(2):145-58.
4
Deoxycholate and cholate modulate the source of cholesterol substrate for bile acid synthesis in the rat.脱氧胆酸盐和胆酸盐调节大鼠胆汁酸合成中胆固醇底物的来源。
Hepatology. 1995 Feb;21(2):529-38.
5
Effect of cholestyramine on the synthesis-ratio of cholic- and chenodeoxycholic acid in hamsters.消胆胺对仓鼠胆酸和鹅去氧胆酸合成比例的影响。
Int Z Klin Pharmakol Ther Toxikol. 1971 Jun;4(4):424-8.
6
Bile acid biosynthesis. Pathways and regulation.胆汁酸生物合成。途径与调控。
Am J Dig Dis. 1974 Oct;19(10):920-9. doi: 10.1007/BF01076215.
7
Bile acid synthesis in the isolated, perfused rabbit liver.离体灌注兔肝脏中的胆汁酸合成
J Clin Invest. 1971 Aug;50(8):1720-30. doi: 10.1172/JCI106661.
8
The role of the pituitary in control of cholesterol 7 -hydroxylase activity in the rat liver.
Hoppe Seylers Z Physiol Chem. 1972 Jul;353(7):1187-8.
9
Hepatic synthesis of bile acids. Biochemical steps and mechanisms of rate control.肝脏中胆汁酸的合成。生化步骤及速率控制机制。
Arch Intern Med. 1972 Oct;130(4):478-87.
10
The regulation of bile acid synthesis in cultured hamster hepatocytes.培养的仓鼠肝细胞中胆汁酸合成的调节
Biochem Soc Trans. 1993 Aug;21 ( Pt 3)(3):284S. doi: 10.1042/bst021284s.

引用本文的文献

1
Effects of ursodiol or ibuprofen on contraction of gallbladder and bile among obese patients during weight loss.熊去氧胆酸或布洛芬对肥胖患者减肥期间胆囊收缩及胆汁的影响。
Dig Dis Sci. 1996 Feb;41(2):242-9. doi: 10.1007/BF02093811.
2
Increased bile acid concentration in liver tissue with cholesterol gallstone disease.胆固醇结石病患者肝脏组织中胆汁酸浓度升高。
J Gastroenterol. 1995 Feb;30(1):61-6. doi: 10.1007/BF01211376.
3
Alteration of cholesterol metabolism by 4-O-methylascochlorin in rats.4-O-甲基曲霉氯菌素对大鼠胆固醇代谢的影响。
Lipids. 1981 Jun;16(6):433-8. doi: 10.1007/BF02535011.
4
Chenodeoxycholic acid: a review of its pharmacological properties and therapeutic use.鹅去氧胆酸：其药理特性与治疗用途综述
Drugs. 1981 Feb;21(2):90-119. doi: 10.2165/00003495-198121020-00002.
5
Effect of bile acid feeding on hepatic steroid 12 alpha-hydroxylase activity in hamsters.胆汁酸喂养对仓鼠肝脏类固醇12α-羟化酶活性的影响。
Lipids. 1983 Nov;18(11):789-94. doi: 10.1007/BF02534637.
6
The present position concerning gallstone dissolution.关于胆结石溶解的现状。
Gut. 1974 Nov;15(11):913-29. doi: 10.1136/gut.15.11.913.
7
The goose that laid the golden bile: gallstone dissolution in man with chenodeoxycholic acid.产金胆汁的鹅：用鹅去氧胆酸溶解人体胆结石
Ir J Med Sci. 1974 Jul;0(0):suppl:115-27. doi: 10.1007/BF02938133.
8
Effect of glucose administration on cholesterol and bile acid metabolism in rats.葡萄糖给药对大鼠胆固醇和胆汁酸代谢的影响。
Lipids. 1975 Aug;10(8):473-7. doi: 10.1007/BF02532431.
9
Effect of phenobarbital, spironolactone and pregnenolone-16 alpha-carbonitrile on bile formation in the rat.苯巴比妥、螺内酯和孕烯醇酮-16α-腈对大鼠胆汁生成的影响。
Naunyn Schmiedebergs Arch Pharmacol. 1975;287(1):33-45. doi: 10.1007/BF00632636.
10
Can colonic bacterial metabolites predispose to cholesterol gall stones?结肠细菌代谢产物会引发胆固醇胆结石吗？
Br Med J. 1975 Feb 22;1(5955):438-40. doi: 10.1136/bmj.1.5955.438.