Angelov A
Vutr Boles. 1979;18(3):214-8.
All deceased with ischemic heart disease (IHD) -- 450 cases and with embolism and thrombosis in pulmonary circulation (ETPC) -- 601 cases were purposefully examined for a period of 10 years (1963--1972). In 75 of the examined a combination of IHD and ETPC was present. In 41 out of them only cicatrices of past infarctions were found and in 34 -- recent myocardial infarctions. In 14 of the examined (41%) it was established, clinically and morphologically, that ETPC are with a longer duration than the recent myocardial infarctions and very likely have played a role in their pathogenesis. The confirmation of acute ischemic changes in myocardium gives a support -- undulation of the myofibrils and fuchsinophylic necrosis in the deceased of ETPC without preceeding IHD. The cardiac hypertrophy and coronary atherosclerosis lead to an intensification of the myocardial ischemic alterations in ETPC.
对所有死于缺血性心脏病(IHD)的450例患者以及死于肺循环栓塞和血栓形成(ETPC)的601例患者进行了为期10年(1963 - 1972年)的专门检查。在接受检查的患者中,有75例同时存在IHD和ETPC。其中41例仅发现既往梗死的瘢痕,34例发现近期心肌梗死。在接受检查的14例患者(41%)中,临床和形态学证实ETPC的病程比近期心肌梗死更长,且很可能在其发病机制中起了作用。在无先前IHD的ETPC死者中,心肌肌原纤维波动和嗜品红坏死等急性缺血改变的证实提供了支持。心脏肥大和冠状动脉粥样硬化导致ETPC中心肌缺血改变加剧。