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公牛精囊炎的研究。II. 盆腔生殖器官畸形作为精囊炎发病机制中可能的诱发因素。

Studies on seminal vesiculitis in the bull. II. Malformation of the pelvic genital organs as a possible predisposing factor in the pathogenesis of seminal vesiculitis.

作者信息

Blom E

出版信息

Nord Vet Med. 1979 Jun;31(6):241-50.

PMID:482093
Abstract

Of 32 bulls showing inflammatory conditions in the pelvic genital organs, 25 were examined post mortem (24 showing vesiculitis with or without ampullitis and one showing ampullitis alone). In ten cases (40%) the following malformations or rare anatomical deviations were demonstrated by careful gross-anatomical dissection: Segmental aplasia of the Wolffian duct: 1; segmental hypoplasia of the Wolffian duct: 3; asymmetric implantation of the two ampullae: 3; accessory vesicular glands: 1; uterus masculinus persistens: 2. These results corroborate earlier observations that a congenital defect may play a certain rôle in the pathogenesis of seminal vesiculitis, most probably by acting as a locus minoris resistentiae in or around colliculus seminalis. On account of the above findings, and considering the prognosis for the condition and the lack of effective treatment, it can in general be recommended, that diseased bulls should be slaughtered as soon as the diagnosis seminal vesiculitis is final.

摘要

在32头出现盆腔生殖器官炎症的公牛中,25头进行了尸检(24头显示有精囊炎伴或不伴有壶腹炎,1头仅显示壶腹炎)。通过仔细的大体解剖,在10例(40%)中发现了以下畸形或罕见的解剖学变异:沃尔夫管节段性发育不全:1例;沃尔夫管节段性发育不良:3例;两个壶腹不对称植入:3例;副精囊腺:1例;永存雄性子宫:2例。这些结果证实了早期的观察结果,即先天性缺陷可能在精囊炎的发病机制中起一定作用,很可能是作为精阜内或其周围的一个抵抗力较弱的部位。鉴于上述发现,并考虑到该病的预后以及缺乏有效的治疗方法,一般建议一旦精囊炎的诊断确定,患病公牛应尽快屠宰。

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