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Biochemical responses of myocardial cells in culture to oxygen and glucose deprivation.

作者信息

DeLuca M A, Ingwall J S, Bittl J A

出版信息

Biochem Biophys Res Commun. 1974 Jul 24;59(2):749-56. doi: 10.1016/s0006-291x(74)80043-4.

DOI:10.1016/s0006-291x(74)80043-4
PMID:4850773
Abstract
摘要

相似文献

1
Biochemical responses of myocardial cells in culture to oxygen and glucose deprivation.
Biochem Biophys Res Commun. 1974 Jul 24;59(2):749-56. doi: 10.1016/s0006-291x(74)80043-4.
2
Influence of variations in pericellular oxygen tension on individual cell growth, muscle-characteristic proteins, and lactate dehydrogenase isoenzyme heart cells.
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Evaluation of dimethylsulfoxide (DMSO) injury to the myocardial cell by determination of high-energy phosphate reserve and enzyme release.
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Factors influencing enzyme release from cultured fetal mouse hearts deprived of oxygen and glucose.影响缺氧和无糖培养的胎鼠心脏释放酶的因素。
J Mol Cell Cardiol. 1978 Oct;10(10):907-19. doi: 10.1016/0022-2828(78)90337-1.
5
Thermally induced myocardial preservation and necrosis in deprived fetal mouse hearts.
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Ischemic myocardial injury in cultured heart cells: leakage of cytoplasmic enzymes from injured cells.培养心肌细胞中的缺血性心肌损伤：受损细胞中细胞质酶的泄漏。
In Vitro. 1978 Aug;14(8):728-32. doi: 10.1007/BF02616170.
7
[Myocardial metabolic condition and enzyme changes in myocardial infarct in relation to residual blood flow].[心肌梗死时心肌代谢状况及酶变化与残余血流的关系]
Langenbecks Arch Chir. 1974;Suppl:11-4.
8
Salient biochemical features in ischemic myocardium.
Circ Res. 1974 Sep;35 Suppl 3:173-81.
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Effect of glucose on enzyme release from, and recovery of, the anoxic myocardium.
Recent Adv Stud Cardiac Struct Metab. 1973;3:763-72.
10
Effect of ischaemia and infarction on the metabolism and function of the isolated, perfused rat heart.缺血和梗死对离体灌注大鼠心脏代谢及功能的影响。
Cardiovasc Res. 1972 May;6(3):284-94. doi: 10.1093/cvr/6.3.284.

引用本文的文献

1
Reduction of cell injury in hypoxic cultures of rat myocardial cells by methylprednisolone.甲基强的松龙减轻大鼠心肌细胞缺氧培养中的细胞损伤。
In Vitro. 1980 Feb;16(2):93-6. doi: 10.1007/BF02831498.
2
Suppression of snake-venom cardiotoxin-induced cardiomyocyte degeneration by blockage of Ca2+ influx or inhibition of non-lysosomal proteinases.通过阻断Ca2+内流或抑制非溶酶体蛋白酶来抑制蛇毒心脏毒素诱导的心肌细胞变性。
Biochem J. 1988 Nov 15;256(1):89-95. doi: 10.1042/bj2560089.
3
Fetal mouse hearts: a model for studying ischemia.胎鼠心脏：一种用于研究缺血的模型。
Proc Natl Acad Sci U S A. 1975 Jul;72(7):2809-13. doi: 10.1073/pnas.72.7.2809.
4
Ischemic myocardial injury in cultured heart cells: leakage of cytoplasmic enzymes from injured cells.培养心肌细胞中的缺血性心肌损伤：受损细胞中细胞质酶的泄漏。
In Vitro. 1978 Aug;14(8):728-32. doi: 10.1007/BF02616170.
5
Ischemic myocardial injury in cultured heart cells: preliminary observations on morphology and beating activity.
In Vitro. 1977;13(12):818-23. doi: 10.1007/BF02615129.