通过传入性迷走神经刺激对迷走神经逃逸现象的调节。
Modulation of vagus escape phenomenon by afferent vagal stimulation.
作者信息
Mohan M, Rai U C
出版信息
Indian J Physiol Pharmacol. 1979 Apr-Jun;23(2):110-4.
The present study was conducted on healthy adult mongrel dogs under chloralose urethane anaesthesia. After bilateral midcervical vagotomy, peripheral end of either vagus was stimulated continuously. This stimulation produced cardiac asystole followed by escape beats. Initially, the heart rate and arterial pressure were low, but both increased progressively and reached a steady-state level in 1-3 min while stimulation of peripheral vagus continued. During this plateau of vagus escape, stimulation of the central end of either vagus produced elevation of arterial pressure and cardio-acceleration. This modulation of vagus escape phenomenon by afferent vagal stimulation was reproducible.
本研究在水合氯醛乌拉坦麻醉下的健康成年杂种犬身上进行。双侧颈中段迷走神经切断术后，持续刺激任一迷走神经的外周端。这种刺激导致心脏停搏，随后出现逸搏。最初，心率和动脉压较低，但在持续刺激外周迷走神经的过程中，两者均逐渐升高，并在1 - 3分钟内达到稳态水平。在迷走神经逸搏的这个平台期，刺激任一迷走神经的中枢端会导致动脉压升高和心率加快。迷走神经传入刺激对迷走神经逸搏现象的这种调节是可重复的。
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