Reversion of the transformed phenotype to the parental phenotype by subcultivation of X-ray-transformed C3H/10T1/2 cells at low cell density.

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作者信息

Brouty-Boyé D, Gresser I, Baldwin C

出版信息

Int J Cancer. 1979 Aug;24(2):253-60. doi: 10.1002/ijc.2910240218.

DOI:10.1002/ijc.2910240218

PMID:489166

Abstract

摘要

相似文献

Reversion of the transformed phenotype to the parental phenotype by subcultivation of X-ray-transformed C3H/10T1/2 cells at low cell density.

Int J Cancer. 1979 Aug;24(2):253-60. doi: 10.1002/ijc.2910240218.

Stability of the phenotypic reversion of x-ray transformed C3H/10T1/2 cells depends on cellular proliferation after subcultivation at low cell density.X射线转化的C3H/10T1/2细胞表型回复的稳定性取决于在低细胞密度下传代培养后的细胞增殖。

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Reversibility of the transformed and neoplastic phenotype. III. Long-term treatment with electrophoretically pure mouse interferon leads to the progressive reversion of the phenotype of X-ray transformed C3H/10T1/2 cells.

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Reversibility of the transformed and neoplastic phenotype. I. Progressive reversion of the phenotype of X-ray-transformed C3H/10T1/2 cells under prolonged treatment with interferon.转化和肿瘤表型的可逆性。I. 用干扰素长期处理下X射线转化的C3H/10T1/2细胞表型的渐进性逆转

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Decreased sensitivity to interferon associated with in vitro transformation of X-ray-transformed C3H/10T1/2 cells.

Int J Cancer. 1979 Aug;24(2):261-5. doi: 10.1002/ijc.2910240219.

Oncogenes in X-ray-transformed C3H 10T1/2 mouse cells and in X-ray-induced mouse fibrosarcoma (RIF-1) cells.X射线转化的C3H 10T1/2小鼠细胞和X射线诱导的小鼠纤维肉瘤（RIF-1）细胞中的癌基因。

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Association of phenotypic reversion of transformed cells induced by interferon with morphological and biochemical changes in the cytoskeleton.干扰素诱导的转化细胞表型逆转与细胞骨架形态学及生化改变的关联

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Enhancement of X-ray-induced transformation in C3H/10T1/2 cells by interferon.

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Sarcomas routinely produced from putatively nontumorigenic Balb/3T3 and C3H/10T1/2 cells by subcutaneous inoculation attached to plastic platelets.

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