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The binding of thrombin by fibrin.

作者信息

Liu C Y, Nossel H L, Kaplan K L

出版信息

J Biol Chem. 1979 Oct 25;254(20):10421-5.

PMID:489603
Abstract
摘要

相似文献

1
The binding of thrombin by fibrin.凝血酶与纤维蛋白的结合。
J Biol Chem. 1979 Oct 25;254(20):10421-5.
2
Unstimulated and thrombin-stimulated platelets binding to immobilized fibrinogen and fibrin on polystyrene supports.未刺激的和凝血酶刺激的血小板与固定在聚苯乙烯载体上的纤维蛋白原和纤维蛋白的结合。
Haemostasis. 1995 Jul-Aug;25(4):158-65. doi: 10.1159/000217156.
3
Regulation of thrombin cleavage of plasma factor XIII bound to fibrin.与纤维蛋白结合的血浆因子 XIII 的凝血酶裂解调节
Ann N Y Acad Sci. 1986;485:140-3. doi: 10.1111/j.1749-6632.1986.tb34576.x.
4
The interaction of thrombin with blood platelets.凝血酶与血小板的相互作用。
Platelets. 2005 Nov;16(7):373-85. doi: 10.1080/09537100500123568.
5
Potentiation of the thrombin induced platelet release reaction by fibrin.
Thromb Res. 1976 Aug;9(2):181-90. doi: 10.1016/0049-3848(76)90162-6.
6
The effect of fibrin clots and clot-bound thrombin on the development of platelet procoagulant activity.纤维蛋白凝块和凝块结合凝血酶对血小板促凝活性发展的影响。
Thromb Haemost. 1995 Sep;74(3):962-8.
7
Phosphatidylinositol 4,5-bisphosphate is selectively retained by platelet-fibrin clots formed by thrombin.磷脂酰肌醇4,5 - 二磷酸被凝血酶形成的血小板 - 纤维蛋白凝块选择性保留。
Biochem J. 1987 Aug 1;245(3):649-53. doi: 10.1042/bj2450649.
8
Interactions of factor XIII with fibrin as substrate and cofactor.因子 XIII 与作为底物和辅因子的纤维蛋白之间的相互作用。
Biochemistry. 1992 Jan 21;31(2):423-9. doi: 10.1021/bi00117a017.
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A role for platelets and thrombin in the juvenile stroke of two siblings with defective thrombin-adsorbing capacity of fibrin(ogen).
Arterioscler Thromb. 1991 Jul-Aug;11(4):785-96. doi: 10.1161/01.atv.11.4.785.
10
The hypersensitivity to thrombin of platelets from diabetic rats is not due to increased thrombin binding.糖尿病大鼠血小板对凝血酶的超敏反应并非由于凝血酶结合增加所致。
Thromb Res. 1991 Feb 15;61(4):469-75. doi: 10.1016/0049-3848(91)90661-f.

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A Mathematical Model of Bivalent Binding Suggests Physical Trapping of Thrombin within Fibrin Fibers.二价结合的数学模型表明凝血酶在纤维蛋白纤维内被物理捕获。
Biophys J. 2019 Oct 15;117(8):1442-1455. doi: 10.1016/j.bpj.2019.09.003. Epub 2019 Sep 13.
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c.259A>C in the fibrinogen gene of alpha chain () is a fibrinogen with thrombotic phenotype.
α链纤维蛋白原基因中的c.259A>C是一种具有血栓形成表型的纤维蛋白原。
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Establishing the Transient Mass Balance of Thrombosis: From Tissue Factor to Thrombin to Fibrin Under Venous Flow.建立血栓的瞬态质量平衡:从组织因子到凝血酶到静脉血流下的纤维蛋白。
Arterioscler Thromb Vasc Biol. 2018 Jul;38(7):1528-1536. doi: 10.1161/ATVBAHA.118.310906. Epub 2018 May 3.
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Coarse-grained molecular dynamics simulations of fibrin polymerization: effects of thrombin concentration on fibrin clot structure.纤维蛋白聚合的粗粒度分子动力学模拟:凝血酶浓度对纤维蛋白凝块结构的影响
J Mol Model. 2018 Apr 5;24(5):109. doi: 10.1007/s00894-018-3642-7.
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Aggregation of thrombin-derived C-terminal fragments as a previously undisclosed host defense mechanism.凝血酶衍生 C 末端片段的聚集作为一种先前未被发现的宿主防御机制。
Proc Natl Acad Sci U S A. 2017 May 23;114(21):E4213-E4222. doi: 10.1073/pnas.1619609114. Epub 2017 May 4.
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Probing the Dynamics of Clot-Bound Thrombin at Venous Shear Rates.探究静脉剪切速率下凝块结合凝血酶的动力学
Biophys J. 2017 Apr 25;112(8):1634-1644. doi: 10.1016/j.bpj.2017.03.002.
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Protease-Activated Receptor-1 Supports Locomotor Recovery by Biased Agonist Activated Protein C after Contusive Spinal Cord Injury.蛋白酶激活受体-1在脊髓挫伤性损伤后通过偏向性激动剂激活蛋白C支持运动功能恢复。
PLoS One. 2017 Jan 25;12(1):e0170512. doi: 10.1371/journal.pone.0170512. eCollection 2017.
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A novel mutation in exon 2 of FGB caused by c.221G>T substitution, predicting the replacement of the native Arginine at position 74 with a Leucine (p.Arg74Leu ) in a proband from a Kurdish family with dysfibrinogenaemia and familial venous and arterial thrombosis.在一个患有异常纤维蛋白原血症以及家族性静脉和动脉血栓形成的库尔德家庭的先证者中,FGB基因第2外显子发生了由c.221G>T替换导致的新型突变,预测第74位天然精氨酸被亮氨酸取代(p.Arg74Leu)。
J Thromb Thrombolysis. 2017 Feb;43(2):263-270. doi: 10.1007/s11239-016-1439-z.