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The pathogenesis of dying-back polyneuropathies. I. An ultrastructural study of experimental tri-ortho-cresyl phosphate intoxication in the cat.

作者信息

Prineas J

出版信息

J Neuropathol Exp Neurol. 1969 Oct;28(4):571-97. doi: 10.1097/00005072-196910000-00003.

DOI:10.1097/00005072-196910000-00003
PMID:4899243
Abstract
摘要

相似文献

1
The pathogenesis of dying-back polyneuropathies. I. An ultrastructural study of experimental tri-ortho-cresyl phosphate intoxication in the cat.
J Neuropathol Exp Neurol. 1969 Oct;28(4):571-97. doi: 10.1097/00005072-196910000-00003.
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The pathogenesis of dying-back polyneuropathies. II. An ultrastructural study of experimental acrylamide intoxication in the cat.逆行性多神经病的发病机制。II. 猫实验性丙烯酰胺中毒的超微结构研究。
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[Phagocytic function of Schwann cells].
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[Submicroscopic changes in myelin in experimental allergic polyneuritis during the process of demyelinization].[实验性变应性多神经炎脱髓鞘过程中髓鞘的亚微观变化]
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9
[Histologic patterns of liver damage in chronic poisoning from tri-ortho-cresyl phosphate (author's transl)].
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Ultrastructure of tri-ortho-cresyl phosphate poisoning in the chicken. II. Studies on spinal cord alterations.鸡磷酸三邻甲苯酯中毒的超微结构。II. 脊髓改变的研究。
Acta Neuropathol. 1970;15(2):142-55. doi: 10.1007/BF00685267.

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Neurochem Res. 1994 Apr;19(4):435-44. doi: 10.1007/BF00967321.
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Acta Neuropathol. 1980;52(2):111-8. doi: 10.1007/BF00688008.
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Arch Toxicol. 1980 Feb;43(4):305-15. doi: 10.1007/BF00366186.
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