Renal tubular acidosis in salt-losing syndromes of congenital adrenal hyperplasia (CAH).
作者信息
Oetliker O H, Zurbrügg R R
出版信息
J Clin Endocrinol Metab. 1970 Oct;31(4):447-50. doi: 10.1210/jcem-31-4-447.
PMID:4916545
Abstract
摘要
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Renal tubular acidosis in salt-losing syndromes of congenital adrenal hyperplasia (CAH).先天性肾上腺增生症（CAH）失盐综合征中的肾小管酸中毒。
J Clin Endocrinol Metab. 1970 Oct;31(4):447-50. doi: 10.1210/jcem-31-4-447.
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Renal tubular acidosis.肾小管酸中毒
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[Astrup's capillary blood test and urinary pH as parameters of renal hydrogen-ion elimination. 2. Deviation of acid-base equilibrium and urinary pH following ammonium chloride loading test and their relationships to latent tubular acidosis].[以阿斯特鲁普毛细血管血液检测和尿液pH值作为肾脏排氢离子参数。2. 氯化铵负荷试验后酸碱平衡和尿液pH值的偏差及其与潜在肾小管酸中毒的关系]
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Renal regulation of fluid, electrolyte, and acid-base homeostasis in the salt-losing syndrome of congenital adrenal hyperplasia (SL-CAH). ECF volume: a compensating factor in aldosterone deficiency.先天性肾上腺增生症失盐综合征（SL-CAH）中肾脏对液体、电解质及酸碱平衡的调节。细胞外液量：醛固酮缺乏时的一个代偿因素。
J Clin Endocrinol Metab. 1978 Apr;46(4):543-51. doi: 10.1210/jcem-46-4-543.
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Congenital adrenal hyperplasia due to combined 21- and 11 beta-hydroxylase deficiency.由21-羟化酶和11β-羟化酶联合缺乏所致的先天性肾上腺增生症。
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