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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Cuthbert A W, Wong P Y

J Physiol. 1971 Dec;219(1):39-56. doi: 10.1113/jphysiol.1971.sp009648.

The sodium-dependent oxygen consumption of pieces of toad bladder (Bufo marinus) has been investigated using an oxygen electrode.2. The effect of polyvalent cations (Ca(2+), Sr(2+), Mg(2+), Eu(3+), La(3+) and Mn(2+)) on sodium-dependent oxygen consumption has been measured. All cations inhibited oxygen consumption, the order of effectiveness being Ca(2+) > Sr(2+) > Mg(2+) > Mn(2+) > Eu(3+) > La(3+).3. Treatment of bladder pieces with antidiuretic hormone (50 m-u./ml.) decreased the effectiveness of Ca(2+) and Sr(2+) as inhibitors of sodium-dependent oxygen consumption. Mn(2+), Eu(2+) and La(2+) were more effective after hormonal treatment, while the effectiveness of Mg(2+) was unaltered.4. The results have been interpreted in terms of a model in which sodium entry to the transporting mechanisms of the epithelium is controlled by Ca(2+), and in which antidiuretic hormone alters Ca(2+) binding and so affects sodium transport.

已使用氧电极研究了海蟾蜍（Bufo marinus）膀胱组织的钠依赖性氧消耗。
已测定了多价阳离子（Ca(2+)、Sr(2+)、Mg(2+)、Eu(3+)、La(3+)和Mn(2+)）对钠依赖性氧消耗的影响。所有阳离子均抑制氧消耗，其有效性顺序为Ca(2+) > Sr(2+) > Mg(2+) > Mn(2+) > Eu(3+) > La(3+)。
用抗利尿激素（50 m-u./ml.）处理膀胱组织降低了Ca(2+)和Sr(2+)作为钠依赖性氧消耗抑制剂的有效性。激素处理后，Mn(2+)、Eu(2+)和La(2+)的有效性更高，而Mg(2+)的有效性未改变。
已根据一个模型对结果进行了解释，在该模型中，钠进入上皮运输机制受Ca(2+)控制，抗利尿激素改变Ca(2+)结合从而影响钠运输。

Cuthbert A W, Wong P Y

J Physiol. 1971 Dec;219(1):39-56. doi: 10.1113/jphysiol.1971.sp009648.

The sodium-dependent oxygen consumption of pieces of toad bladder (Bufo marinus) has been investigated using an oxygen electrode.2. The effect of polyvalent cations (Ca(2+), Sr(2+), Mg(2+), Eu(3+), La(3+) and Mn(2+)) on sodium-dependent oxygen consumption has been measured. All cations inhibited oxygen consumption, the order of effectiveness being Ca(2+) > Sr(2+) > Mg(2+) > Mn(2+) > Eu(3+) > La(3+).3. Treatment of bladder pieces with antidiuretic hormone (50 m-u./ml.) decreased the effectiveness of Ca(2+) and Sr(2+) as inhibitors of sodium-dependent oxygen consumption. Mn(2+), Eu(2+) and La(2+) were more effective after hormonal treatment, while the effectiveness of Mg(2+) was unaltered.4. The results have been interpreted in terms of a model in which sodium entry to the transporting mechanisms of the epithelium is controlled by Ca(2+), and in which antidiuretic hormone alters Ca(2+) binding and so affects sodium transport.

已使用氧电极研究了海蟾蜍（Bufo marinus）膀胱组织的钠依赖性氧消耗。
已测定了多价阳离子（Ca(2+)、Sr(2+)、Mg(2+)、Eu(3+)、La(3+)和Mn(2+)）对钠依赖性氧消耗的影响。所有阳离子均抑制氧消耗，其有效性顺序为Ca(2+) > Sr(2+) > Mg(2+) > Mn(2+) > Eu(3+) > La(3+)。
用抗利尿激素（50 m-u./ml.）处理膀胱组织降低了Ca(2+)和Sr(2+)作为钠依赖性氧消耗抑制剂的有效性。激素处理后，Mn(2+)、Eu(2+)和La(2+)的有效性更高，而Mg(2+)的有效性未改变。
已根据一个模型对结果进行了解释，在该模型中，钠进入上皮运输机制受Ca(2+)控制，抗利尿激素改变Ca(2+)结合从而影响钠运输。