Chesson A L, Schochet S S, Peters B H
Arch Neurol. 1979 Nov;36(11):700-4. doi: 10.1001/archneur.1979.00500470070014.
A young man with episodes characteristic of familial normokalemic periodic paralysis was restudied after a change in the nature of his attacks. He now has spontaneously occurring and provokable episodes of both hyperkalemic and hypokalemic periodic paralyses that are superimposed on a persistent myopathy. Hypokalemia seems to be associated with facilitated entry of glucose and potassium into muscles, whereas resistance to glucose and potassium entry accompanies hyperkalemia. The possibility that episodes of both hypokalemic and hyperkalemic weaknesses can occur in one person should be considered in pathogenic postulates regarding periodic paralysis.
一名具有家族性正常血钾性周期性麻痹特征发作的年轻男子，在其发作性质改变后接受了再次研究。他现在既有自发性的，也可诱发性的高血钾性和低血钾性周期性麻痹发作，且叠加在持续性肌病之上。低血钾似乎与葡萄糖和钾更容易进入肌肉有关，而高血钾时则伴有对葡萄糖和钾进入的抵抗。在关于周期性麻痹的发病机制假设中，应考虑一个人可能同时出现低血钾性和高血钾性肌无力发作的可能性。
