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Phenylacetic acid as a potential therapeutic agent for the treatment of human cancer.

作者信息

Neish W J

出版信息

Experientia. 1971 Jul;27(7):860-1. doi: 10.1007/BF02136914.

DOI:10.1007/BF02136914
PMID:5139518
Abstract
摘要

相似文献

1
Phenylacetic acid as a potential therapeutic agent for the treatment of human cancer.
Experientia. 1971 Jul;27(7):860-1. doi: 10.1007/BF02136914.
2
Inhibition of Rd/3 rat sarcoma by L-asparaginase alone and in combination with sodium para-amino-salicylate.单独使用L-天冬酰胺酶以及联合对氨基水杨酸钠对Rd/3大鼠肉瘤的抑制作用。
Z Krebsforsch Klin Onkol Cancer Res Clin Oncol. 1973;79(2):78-84. doi: 10.1007/BF00284381.
3
Total parenteral nutrition, glutamine, and tumor growth.全胃肠外营养、谷氨酰胺与肿瘤生长
JPEN J Parenter Enteral Nutr. 1990 Jul-Aug;14(4 Suppl):86S-89S. doi: 10.1177/0148607190014004101.
4
Increased whole-body protein and glutamine turnover in advanced cancer is not matched by an increased muscle protein and glutamine turnover.晚期癌症患者全身蛋白质和谷氨酰胺周转率增加，但肌肉蛋白质和谷氨酰胺周转率并未相应增加。
J Surg Res. 1997 Feb 15;68(1):44-55. doi: 10.1006/jsre.1997.5007.
5
[Effect of alkoxybenzylpyrimidylamidophosphoric acid diethyleneimides on DNA and RNA biosynthesis in the tissues of tumor-bearing rats and in sarcoma-45].
Zh Eksp Klin Med. 1979;19(3):66-71.
6
[Antineoplastic and antimitotic effect of prospidin in hypophysectomized and thyroidectomized rats with sarcoma 45].
Farmakol Toksikol. 1974 Nov-Dec;37(6):715-7.
7
[Effect of an active carcinolytic on the level of DNA methylation of several tissues of intact and tumor-bearing rats].[活性溶癌物质对完整大鼠和荷瘤大鼠多种组织DNA甲基化水平的影响]
Zh Eksp Klin Med. 1977;17(4):42-6.
8
Can tumor oxygenation be improved by reducing cellular oxygen consumption.
Adv Exp Med Biol. 1999;471:525-32. doi: 10.1007/978-1-4615-4717-4_62.
9
[Effect of thiophosphamide on the incorporation of H3-thymidine into the DNA of cells of surviving transplantable tumor sections sensitive and resistant toward the agent].[硫代磷酰胺对存活的对该药物敏感和耐药的可移植肿瘤切片细胞DNA中H3-胸腺嘧啶核苷掺入的影响]
Farmakol Toksikol. 1971;34(1):91-7.
10
[Testing of cytostatic agents on individual tumors].[细胞抑制剂对个体肿瘤的测试]
Acta Histochem. 1968:Suppl 8:331+.

引用本文的文献

1
Aromatic amino acids play a harmonizing role in prostate cancer: A metabolomics-based cross-sectional study.芳香族氨基酸在前列腺癌中发挥协调作用：一项基于代谢组学的横断面研究。
Int J Reprod Biomed. 2021 Sep 9;19(8):741-750. doi: 10.18502/ijrm.v19i8.9622. eCollection 2021 Aug.
2
Synergistic inhibition of human rhabdomyosarcoma cells by sodium phenylacetate and Tretinoin.
In Vitro Cell Dev Biol. 1993 Mar;29A(3 Pt 1):189-91. doi: 10.1007/BF02634180.
3
Inhibition of Rd/3 rat sarcoma by L-asparaginase alone and in combination with sodium para-amino-salicylate.单独使用L-天冬酰胺酶以及联合对氨基水杨酸钠对Rd/3大鼠肉瘤的抑制作用。

本文引用的文献

1
Chemical characteristics and physiological roles of glutamine.谷氨酰胺的化学特性与生理作用
Chem Rev. 1945 Oct;37:161-208. doi: 10.1021/cr60117a001.
2
Lethal effect of indolyl-3-acetic acid ethyl ester and protective effect of indolyl-3-acetic acid on free cells of the Ehrlich ascites carcinoma.吲哚 - 3 - 乙酸乙酯对艾氏腹水癌细胞游离细胞的致死作用及吲哚 - 3 - 乙酸的保护作用
Nature. 1963 May 11;198:553-4. doi: 10.1038/198553a0.
3
Synthetic auxins in the chemotherapy of transplanted and spontaneous mouse adenocarcinoma.合成生长素在移植性和自发性小鼠腺癌化疗中的应用
Z Krebsforsch Klin Onkol Cancer Res Clin Oncol. 1973;79(2):78-84. doi: 10.1007/BF00284381.
J Natl Cancer Inst. 1960 Nov;25:1111-20.
4
[Inhibition of respiration by means of carcinostatically active substances].
Z Krebsforsch. 1959;62(6):653-7.
5
Specific rotation of phenylacetylglutamine excreted by cancer patients and normal subjects.癌症患者和正常受试者排泄的苯乙酰谷氨酰胺的比旋度。
Experientia. 1958 Jul 15;14(7):247-8. doi: 10.1007/BF02159184.
6
Synthesis of phenylacetylglutamine by human tissue.人体组织合成苯乙酰谷氨酰胺。
J Biol Chem. 1957 Nov;229(1):463-76.
7
The hypoglycemic and insulinase-inhibitory action of some plant growth regulators.某些植物生长调节剂的降血糖和胰岛素酶抑制作用。
Endocrinology. 1956 Dec;59(6):715-8. doi: 10.1210/endo-59-6-715.
8
[The effect of substituted diketones and esters on the growth of Walker carcinoma].
Z Krebsforsch. 1956;61(1):78-80.
9
Metabolism of glutamine.谷氨酰胺的代谢
Physiol Rev. 1956 Jan;36(1):103-27. doi: 10.1152/physrev.1956.36.1.103.