Meyer T, Hüsch M, van den Berg E, Ködding R, Höffken B, Hesch R D
Dtsch Med Wochenschr. 1979 Nov 30;104(48):1711-4. doi: 10.1055/s-0028-1129177.
Triiodothyronine (T3) was administered to 11 patients in septic shock. In all there occurred an increase in arterial blood pressure within 24 hours, the rise in systolic pressure being 34 +/- 4.2 mm Hg, of diastolic 14 +/- 8.2 mm Hg, corresponding to a rise in mean arterial pressure of 25 +/- 6.1 mm Hg. Haemodynamic studies in four patients suggest that the action of T3 is mediated via beta-sympathomimetic channels.
对11名感染性休克患者给予三碘甲状腺原氨酸(T3)。所有患者在24小时内动脉血压均升高,收缩压升高34±4.2毫米汞柱,舒张压升高14±8.2毫米汞柱,平均动脉压升高25±6.1毫米汞柱。对4名患者的血流动力学研究表明,T3的作用是通过β-拟交感神经通道介导的。