胆固醇酯代谢 - Suppr

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Cholesterol ester metabolism.

作者信息

Goodman D S

出版信息

Physiol Rev. 1965 Oct;45(4):747-839. doi: 10.1152/physrev.1965.45.4.747.

DOI:10.1152/physrev.1965.45.4.747

PMID:5318997

Abstract

摘要

相似文献

Cholesterol ester metabolism.胆固醇酯代谢

Physiol Rev. 1965 Oct;45(4):747-839. doi: 10.1152/physrev.1965.45.4.747.

[Lipids of brain tumors].[脑肿瘤的脂质]

Dtsch Z Nervenheilkd. 1965 Dec 14;187(8):737-48.

Regulation of cholesterol metabolism in rat adrenal glands: effect of adrenocorticotropin, cholesterol, and corticosteroids on acyl-coenzyme A synthetase and cholesterol ester hydrolases.

Endocrinology. 1984 Jan;114(1):136-40. doi: 10.1210/endo-114-1-136.

Studies on the importance of lecithin for cholesterol solubilization in bile.关于卵磷脂在胆汁中胆固醇溶解作用重要性的研究。

J Lab Clin Med. 1966 Jul;68(1):90-102.

[Effect of ACTH on some aspects of cholesterol and cholesterol ester metabolism in the rat's adrenal glands].

Probl Endokrinol (Mosk). 1973 Sep-Oct;19(5):67-72.

Formation of cholesteryl ester-rich particulate lipid during metabolism of chylomicrons.乳糜微粒代谢过程中富含胆固醇酯的颗粒脂质的形成。

J Clin Invest. 1970 Mar;49(3):465-71. doi: 10.1172/JCI106255.

Regulation of ovarian cholesterol metabolism: control of 3-hydroxy-3-methylglutaryl coenzyme A reductase and acyl coenzyme A:cholesterol acyltransferase.卵巢胆固醇代谢的调节：3-羟基-3-甲基戊二酰辅酶A还原酶及酰基辅酶A：胆固醇酰基转移酶的调控

Endocrinology. 1981 Apr;108(4):1476-86. doi: 10.1210/endo-108-4-1476.

[Cholesterol ester transfer protein. A major player in cholesterol metabolism].[胆固醇酯转运蛋白。胆固醇代谢的主要参与者]

Ugeskr Laeger. 1999 Sep 20;161(38):5295-8.

[Proceedings: 140. Effect of nicotinic acid ester derivative on cholesterol metabolism (author's transl)].[会议论文集：140. 烟酸酯衍生物对胆固醇代谢的影响（作者译）]

Nihon Seirigaku Zasshi. 1973 Aug-Sep;35(8):441.

Cholesterol ester metabolism in the brain: properties and subcellular distribution of cholesterol-esterifying enzymes and cholesterol ester hydrolases in adult rat brain.大脑中的胆固醇酯代谢：成年大鼠大脑中胆固醇酯化酶和胆固醇酯水解酶的特性及亚细胞分布

Biochim Biophys Acta. 1971 Jul 13;239(2):293-311.

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PRD125, a potent and selective inhibitor of sterol O-acyltransferase 2 markedly reduces hepatic cholesteryl ester accumulation and improves liver function in lysosomal acid lipase-deficient mice.PRD125是一种强效且选择性的固醇O-酰基转移酶2抑制剂，可显著减少溶酶体酸性脂肪酶缺陷小鼠的肝脏胆固醇酯积累并改善肝功能。

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Deletion of sterol O-acyltransferase 2 (SOAT2) function in mice deficient in lysosomal acid lipase (LAL) dramatically reduces esterified cholesterol sequestration in the small intestine and liver.在溶酶体酸性脂肪酶（LAL）缺乏的小鼠中，甾醇O-酰基转移酶2（SOAT2）功能的缺失显著降低了小肠和肝脏中酯化胆固醇的蓄积。

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Acute sterol o-acyltransferase 2 (SOAT2) knockdown rapidly mobilizes hepatic cholesterol for fecal excretion.急性固醇O-酰基转移酶2（SOAT2）的敲低可迅速动员肝脏胆固醇用于粪便排泄。

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A systematic survey of lipids across mouse tissues.对小鼠组织中的脂质进行系统调查。

Am J Physiol Endocrinol Metab. 2014 Apr 15;306(8):E854-68. doi: 10.1152/ajpendo.00371.2013. Epub 2014 Feb 11.

Loss of β-carotene 15,15'-oxygenase in developing mouse tissues alters esterification of retinol, cholesterol and diacylglycerols.发育中小鼠组织中β-胡萝卜素15,15'-加氧酶的缺失会改变视黄醇、胆固醇和二酰甘油的酯化作用。

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Tissue-specific knockouts of ACAT2 reveal that intestinal depletion is sufficient to prevent diet-induced cholesterol accumulation in the liver and blood.组织特异性敲除 ACAT2 表明，肠道耗竭足以防止饮食诱导的胆固醇在肝脏和血液中积累。

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Cholesteryl ester hydrolase activity is abolished in HSL-/- macrophages but unchanged in macrophages lacking KIAA1363.胆固醇酯水解酶活性在 HSL-/- 巨噬细胞中被废除，但在缺乏 KIAA1363 的巨噬细胞中不变。

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