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[Etiology of hip dysplasia (attempt at a comparative study)].

作者信息

Rütt A, von Schmoller G

出版信息

Arch Orthop Unfallchir. 1969 Jul;66(3):220-5. doi: 10.1007/BF00416051.

DOI:10.1007/BF00416051
PMID:5347566
Abstract
摘要

相似文献

1
[Etiology of hip dysplasia (attempt at a comparative study)].[髋关节发育不良的病因(一项比较研究的尝试)]
Arch Orthop Unfallchir. 1969 Jul;66(3):220-5. doi: 10.1007/BF00416051.
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On the etiology and pathogenesis of hip dysplasia: a comparative review.
J Small Anim Pract. 1966 Nov;7(11):673-88. doi: 10.1111/j.1748-5827.1966.tb04393.x.
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Asymmetry of the epiphyseal nucleus in the femoral head in stable and unstable hip joints.稳定和不稳定髋关节中股骨头骨骺核的不对称性。
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[Clinico-roentgenographic diagnosis of dysplasia, subluxation and dislocation of the hip in children during the 1st year of life].
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8
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[Etiological relationship of dysplasia of the hip joint and congenital dislocation of the hip].髋关节发育不良与先天性髋关节脱位的病因关系
Ortop Travmatol Protez. 1989 Mar(3):56-8.
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Arch Orthop Unfallchir. 1972;72(3):277-86. doi: 10.1007/BF00416419.

本文引用的文献

1
Pelvic instability in newborns with special reference to congenital dislocation of the hip and hormonal factors. A roentgenologic study.新生儿骨盆不稳定性,特别涉及先天性髋关节脱位和激素因素。一项X线研究。
Acta Radiol Suppl. 1962;212:1-66.
2
Disturbed urinary excretion pattern of oestrogens in newborns with congenital dislocation of the hip. I. The excretion of oestrogen during the first few days of life.先天性髋关节脱位新生儿雌激素的尿排泄模式紊乱。I. 出生后最初几天的雌激素排泄情况。
Acta Endocrinol (Copenh). 1961 Jul;37:423-6. doi: 10.1530/acta.0.0370423.
3
Studies on oestrogen metabolism in new-born boys. I. Excretion of oestrone, oestradiol-17 beta and oestriol during the first few days of life.
新生男婴雌激素代谢的研究。I. 出生后最初几天雌酮、雌二醇-17β和雌三醇的排泄情况。
Acta Endocrinol (Copenh). 1957 Nov;26(3):303-12.
4
Influence of early rapid growth and weight gain on hip dysplasia in the German Shepherd Dog.早期快速生长和体重增加对德国牧羊犬髋关节发育不良的影响。
J Am Vet Med Assoc. 1964 Oct 1;145(7):661-8.
5
On the etiology and pathogenesis of hip dysplasia: a comparative review.
J Small Anim Pract. 1966 Nov;7(11):673-88. doi: 10.1111/j.1748-5827.1966.tb04393.x.