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The reversal of triparanol-induced cataracts in the rat.

作者信息

Harris J E, Gruber L

出版信息

Doc Ophthalmol. 1969;26:324-33. doi: 10.1007/BF00943994.

DOI:10.1007/BF00943994
PMID:5359529
Abstract
摘要

相似文献

1
The reversal of triparanol-induced cataracts in the rat.大鼠中三苯乙醇诱导的白内障的逆转
Doc Ophthalmol. 1969;26:324-33. doi: 10.1007/BF00943994.
2
Lipid composition and (Na+ + K+)-ATPase activity in rat lens during triparanol-induced cataract formation.在三苯乙醇诱导大鼠晶状体形成白内障过程中的脂质组成及（钠+钾）-ATP酶活性
Biochim Biophys Acta. 1981 Jun 9;644(1):1-12. doi: 10.1016/0005-2736(81)90052-3.
3
Reversal of triparanol-induced cataracts in the rat. II. Exchange of 22 Na, 42 K, and 86 Rb in cataractous and clearing lenses.
Invest Ophthalmol. 1972 Jul;11(7):608-16.
4
Cataracts induced by surface active agents.
Exp Eye Res. 1974 Jul;19(1):35-42. doi: 10.1016/0014-4835(74)90069-4.
5
The reversal of triparanol-induced cataract in the rat. IV. Reduced sulfhydryl groups in soluble protein and glutathione.
Invest Ophthalmol. 1973 May;12(5):388-90.
6
[Studies on the enucleated lens. 3. Cation content in naphthalene cataractous lens].[去核晶状体的研究。3. 萘性白内障晶状体中的阳离子含量]
Yakugaku Zasshi. 1969 Jun;89(6):745-9. doi: 10.1248/yakushi1947.89.6_745.
7
Biochemical changes associated with the development and reversal of galactose cataracts.与半乳糖性白内障的发生和逆转相关的生化变化。
Exp Eye Res. 1976 Nov;23(5):483-93. doi: 10.1016/0014-4835(76)90157-3.
8
Tissue-specific inhibition of ATPase activities in rat induced by triparanol feeding.喂食三苯乙醇诱导大鼠组织特异性ATP酶活性抑制。
Exp Eye Res. 1983 Aug;37(2):209-12. doi: 10.1016/0014-4835(83)90080-5.
9
[Studies on the electrolytes and water in the various experimental cataracts. I. Change in electrolytes and water content of the naphthalene cataract].
Nippon Ganka Gakkai Zasshi. 1968 Sep;72(9):1753-8.
10
[Experimental cataract induced by hypertonic solutions].
Nippon Ganka Gakkai Zasshi. 1972 Aug;76(8):718-26.

引用本文的文献

1
Retinal structure and function in an animal model that replicates the biochemical hallmarks of desmosterolosis.一种复制鲨烯醇增多症生化特征的动物模型中的视网膜结构与功能
Neurochem Res. 2000 May;25(5):685-94. doi: 10.1023/a:1007519321917.
2
Distribution and movement of ions in the ocular lens.眼内晶状体中离子的分布与移动。
Doc Ophthalmol. 1972 Apr 15;31(1):1-28. doi: 10.1007/BF00167360.

本文引用的文献

1
Lethal effect in dogs of prolonged triparanol oral administration.长期口服三苯乙醇对犬的致死作用。
Arch Pathol. 1962 Jun;73:445-50.
2
Cataracts in patients treated with triparanol.接受曲帕拉醇治疗的患者中的白内障。
JAMA. 1962 Jul 28;181:339-40. doi: 10.1001/jama.1962.03050300059020a.
3
Cataract formation after triparanol therapy.曲帕拉醇治疗后白内障的形成。
Arch Ophthalmol. 1962 Oct;68:486-9. doi: 10.1001/archopht.1962.00960030490010.
4
TISSUE DISTRIBUTION OF CHOLESTEROL AND 24-DEHYDROCHOLESTEROL DURING CHRONIC TRIPARANOL THERAPY.慢性三苯乙醇治疗期间胆固醇和24-脱氢胆固醇的组织分布
J Lipid Res. 1965 Jan;6:37-42.
5
POINT OF IRREVERSIBILITY OF GALACTOSE CATARACTS IN THE RAT.大鼠半乳糖性白内障的不可逆点
Proc Soc Exp Biol Med. 1965 Feb;118:324-6. doi: 10.3181/00379727-118-29832.
6
HYDRATION OF THE LENS DURING THE DEVELOPMENT OF GALACTOSE CATARACT.半乳糖性白内障形成过程中晶状体的水合作用
Invest Ophthalmol. 1964 Dec;3:577-84.
7
TRIPARANOL INDUCED CATARACT IN RATS.
Arch Ophthalmol. 1963 Oct;70:522-30. doi: 10.1001/archopht.1963.00960050524016.
8
Osmotic changes in experimental galactose cataracts.实验性半乳糖性白内障中的渗透压变化
Exp Eye Res. 1962 Jun;1:405-10. doi: 10.1016/s0014-4835(62)80030-x.
9
Lens opacities produced by cataractogenic agents, with special reference to triparanol cataract.
Am J Ophthalmol. 1963 Mar;55:537-41.
10
The mechanism of action of MER-29.MER-29的作用机制。
Prog Cardiovasc Dis. 1960 May;2:525-30. doi: 10.1016/s0033-0620(60)80024-2.