Nakajima T, Kusumoto S, Oda H, Nogami H
Southeast Asian J Trop Med Public Health. 1979 Dec;10(4):604-9.
The animal studies in mice resulted as follows. Long-term exposure to NO2 at or above 0.5 ppm affected primarily the respiratory organs. The pulmonary effect of NO was slighter than NO2. Nitrosylhemoglobin formation in vivo was much smaller than in vitro strong affinity of NO with hemoglobin. The components other than O3 contained in the photochemically formed oxidant mixtures enhanced the effect of O3 alone. Symptoms of the patients seriously injured by photochemical smog in Japan suggested the different type in quality from the Los Angeles-type smog.
对小鼠的动物研究结果如下。长期暴露于0.5 ppm及以上的二氧化氮主要影响呼吸器官。一氧化氮对肺部的影响比二氧化氮小。体内亚硝基血红蛋白的形成比体外一氧化氮与血红蛋白的强亲和力要小得多。光化学形成的氧化剂混合物中所含的除臭氧以外的成分增强了单独臭氧的作用。日本因光化学烟雾严重受伤患者的症状表明其在质量上与洛杉矶型烟雾不同。
