Yutsudo M
J Gen Virol. 1979 Nov;45(2):351-9. doi: 10.1099/0022-1317-45-2-351.
Two polypeptides (imm-a and imm-b) which are not induced by an immunity mutant T4Dimm2 but by a wild-type strain T4D were identified by sodium dodecyl sulphate (SDS)-polyacrylamide gel electrophoresis. Their mol. wt. were 77 000 and 45 000, respectively. These polypeptides exhibited a similar kinetic pattern of synthesis. Within a few minutes p.i. the primary phage established the system that inhibited imm gene expression of superinfecting phage. This was shown by measuring both the phenotypic expression of immunity and the synthesis of imm gene polypeptides. The expression of two other immediate-early genes, namely genes s and 30, and early gene 33, was not affected by primary infection.
通过十二烷基硫酸钠（SDS）-聚丙烯酰胺凝胶电泳鉴定出两种多肽（免疫-a和免疫-b），它们不是由免疫突变体T4Dimm2诱导产生，而是由野生型菌株T4D诱导产生。它们的分子量分别为77000和45000。这些多肽表现出相似的合成动力学模式。感染后几分钟内，初级噬菌体建立了抑制超感染噬菌体免疫基因表达的系统。这通过测量免疫的表型表达和免疫基因多肽的合成得以证明。另外两个立即早期基因，即基因s和30，以及早期基因33的表达不受初次感染的影响。
