Bautista J, Gil-Neciga E, Gil-Peralta A
Eur Neurol. 1979;18(6):415-20. doi: 10.1159/000115114.
A case of a patient suffering from primary hyperaldosteronism is reported. In this case the disease is manifested clinically by periodic paralysis and hypopotasemia without permanent myopathy. The morphological study of the muscle demonstrates selective atrophy of the type 2A fibers as the most pronounced alteration. These findings suggest a chronic myopathic process.
报告了一例原发性醛固酮增多症患者。在该病例中,疾病临床上表现为周期性麻痹和低钾血症,无永久性肌病。肌肉的形态学研究显示,2A型纤维选择性萎缩是最明显的改变。这些发现提示存在慢性肌病过程。