BIO 14.6 营养不良仓鼠骨骼肌线粒体呼吸及氧化磷酸化的钙相关镁反应缺陷 - Suppr

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A calcium-associated magnesium-responsive defect of respiration and oxidative phosphorylation by skeletal muscle mitochondria of BIO 14.6 dystrophic hamsters.

作者信息

Wrogemann K, Blanchaer M C, Jacobson B E

出版信息

Life Sci II. 1970 Oct 22;9(20):1167-73. doi: 10.1016/0024-3205(70)90035-4.

DOI:10.1016/0024-3205(70)90035-4

PMID:5477798

Abstract

摘要

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A calcium-associated magnesium-responsive defect of respiration and oxidative phosphorylation by skeletal muscle mitochondria of BIO 14.6 dystrophic hamsters.BIO 14.6 营养不良仓鼠骨骼肌线粒体呼吸及氧化磷酸化的钙相关镁反应缺陷

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Increased muscle calcium. A possible cause of mitochondrial dysfunction and cellular necrosis in denervated rat skeletal muscle.肌肉钙含量增加。去神经支配的大鼠骨骼肌中线粒体功能障碍和细胞坏死的一个可能原因。

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The histochemical characterization of the coupling state of skeletal muscle mitochondria.骨骼肌线粒体偶联状态的组织化学特征

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Ultrastructural changes in muscle mitochondria in situ, including the apparent development of internal septa, associated with the uptake and release of calcium.肌肉线粒体在原位的超微结构变化，包括内部隔膜的明显形成，与钙的摄取和释放有关。

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Calcium uptake in skeletal muscle mitochondria. I. The effects of chelating agents on the mitochondria from fatigued rats.骨骼肌线粒体对钙的摄取。I. 螯合剂对疲劳大鼠线粒体的影响。

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