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Polykaryocytosis and mitosis in a human cell line after treatment with measles virus.

作者信息

Heneen W K, Nichols W W, Levan A, Norrby E

出版信息

Hereditas. 1970;64(1):53-84. doi: 10.1111/j.1601-5223.1970.tb02273.x.

DOI:10.1111/j.1601-5223.1970.tb02273.x
PMID:5525758
Abstract
摘要

相似文献

1
Polykaryocytosis and mitosis in a human cell line after treatment with measles virus.麻疹病毒处理后人类细胞系中的多核细胞增多和有丝分裂
Hereditas. 1970;64(1):53-84. doi: 10.1111/j.1601-5223.1970.tb02273.x.
2
Ultrastructure of a measles-carrier human cell line.一株麻疹病毒携带人类细胞系的超微结构
Acta Pathol Microbiol Scand A. 1973 May;81(3):281-90. doi: 10.1111/j.1699-0463.1973.tb03537.x.
3
Relation of virus-induce cell fusion and chromosome pulverization to mitotic events.病毒诱导的细胞融合和染色体粉碎与有丝分裂事件的关系。
J Natl Cancer Inst. 1969 Aug;43(2):335-47.
4
Cytogenetic study of the continuous RH cell line and its clones with a different susceptibility to measles virus.对连续RH细胞系及其对麻疹病毒敏感性不同的克隆进行细胞遗传学研究。
Acta Virol. 1975 May;19(3):204-8.
5
[Mitosis and virus infection in cell cultures. II. Reaction of the nuclear apparatus of the cell to virus infection].[细胞培养中的有丝分裂与病毒感染。II. 细胞的核器对病毒感染的反应]
Usp Sovrem Biol. 1975 May-Jun;79(3):420-8.
6
The mutagenic effect of measles virus on cell cultures of different origin.
Acta Virol. 1975 Jul;19(4):299-304.
7
[Effect of virulent and vaccine strains of measles virus on the chromosomal apparatus of the cell].[麻疹病毒强毒株和疫苗株对细胞染色体装置的影响]
Vopr Virusol. 1972 Sep-Oct;17(5):611-6.
8
Mechanism of early and late polykaryocytosis induced by the bovine leukaemia virus.牛白血病病毒诱导早期和晚期多核细胞增多症的机制。
J Gen Virol. 1981 Nov;57(Pt 1):227-31. doi: 10.1099/0022-1317-57-1-227.
9
Cytogenetic modifications induced in human diploid cells by mixed infection with measles and cytomegalic virus.麻疹病毒与巨细胞病毒混合感染诱导人二倍体细胞的细胞遗传学改变。
Virologie. 1981 Jul-Sep;32(3):199-206.
10
Fusion of LLC-MK2 tissue culture cells induced by measles virus.
Arch Gesamte Virusforsch. 1966;19(2):91-109. doi: 10.1007/BF01241490.

引用本文的文献

1
Animal and human RNA viruses: genetic variability and ability to overcome vaccines.动物和人类 RNA 病毒:遗传变异性和克服疫苗的能力。
Arch Microbiol. 2021 Mar;203(2):443-464. doi: 10.1007/s00203-020-02040-5. Epub 2020 Sep 28.
2
Extensive cytological damage caused by measles in African children.麻疹在非洲儿童中造成的广泛细胞学损伤。
J Med Genet. 1974 Jun;11(2):171-6. doi: 10.1136/jmg.11.2.171.
3
The fluorescence karyotypes of three human meningiomas with hyperdiploid-hypotriploid stemlines.
Acta Neuropathol. 1973 Jun 26;25(1):46-53. doi: 10.1007/BF00686857.
4
Pattern of DNA segregation in multipolar anatelophases of different ploidy in euploid and aneuploid mammalian cells cultivated in vitro.体外培养的整倍体和非整倍体哺乳动物细胞中不同倍性多极后期的DNA分离模式。
Genetica. 1972;43(1):130-47. doi: 10.1007/BF00122506.
5
Studies on leukocyte cultures in measles. II. Detection of measles virus antigen in human leucocytes by immunofluorescence.麻疹患者白细胞培养的研究。II. 用免疫荧光法检测人白细胞中的麻疹病毒抗原。
Arch Gesamte Virusforsch. 1974;44(4):323-9. doi: 10.1007/BF01251013.
6
Studies on leukocyte cultures in measles. I. Lymphocyte transformation and giant cell formation in leukocyte cultures from clinical cases of measles.麻疹白细胞培养的研究。I. 麻疹临床病例白细胞培养中的淋巴细胞转化和巨细胞形成。
Arch Gesamte Virusforsch. 1974;44(4):313-22.
7
Induction of nuclear envelopes around metaphase chromosomes after fusion with interphase cells.与间期细胞融合后在中期染色体周围诱导形成核膜。
J Cell Biol. 1971 Oct;51(1):104-15. doi: 10.1083/jcb.51.1.104.
8
In situ analysis of normal and abnormal patterns of the mitotic apparatus in cultured rat-kangaroo cells.培养的大鼠袋鼠细胞有丝分裂器正常和异常模式的原位分析。
Chromosoma. 1970;29(1):88-117. doi: 10.1007/BF01183663.
9
Euploid segregation through multipolar mitosis in mammalian cell cultures. Identification of triploid, haploid, and segregating diploid cells in a diploid-euploid primary culture of rhesus kidney cells.哺乳动物细胞培养中通过多极有丝分裂实现整倍体分离。在恒河猴肾细胞的二倍体 - 整倍体原代培养物中鉴定三倍体、单倍体和正在分离的二倍体细胞。
Chromosoma. 1974 Mar 14;45(2):151-62. doi: 10.1007/BF00362308.
10
Studies of multipolar mitoses in euploid tissue cultures. I. Somatic reduction to exactly haploid and triploid chromosome sets.整倍体组织培养中多极有丝分裂的研究。I. 体细胞减数分裂为精确的单倍体和三倍体染色体组。
Chromosoma. 1973 May 14;42(1):71-86. doi: 10.1007/BF00326331.