先天性汞中毒 - Suppr

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Congenital mercury poisoning.

作者信息

Snyder R D

出版信息

N Engl J Med. 1971 May 6;284(18):1014-6. doi: 10.1056/NEJM197105062841806.

DOI:10.1056/NEJM197105062841806

PMID:5573294

Abstract

摘要

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引用本文的文献

Post-translational modifications in MeHg-induced neurotoxicity.汞诱导神经毒性的翻译后修饰。

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Total mercury exposure in early pregnancy has no adverse association with scholastic ability of the offspring particularly if the mother eats fish.孕妇在早期接触总汞并不会对后代的学习能力产生不良影响，特别是如果母亲食用鱼类。

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Postnatal Migration of Cerebellar Interneurons.小脑中间神经元的出生后迁移

Brain Sci. 2017 Jun 6;7(6):62. doi: 10.3390/brainsci7060062.

Rescue of neuronal migration deficits in a mouse model of fetal Minamata disease by increasing neuronal Ca2+ spike frequency.通过增加神经元钙峰频率挽救胎儿水俣病小鼠模型中的神经元迁移缺陷

Proc Natl Acad Sci U S A. 2012 Mar 27;109(13):5057-62. doi: 10.1073/pnas.1120747109. Epub 2012 Mar 12.

Methylmercury: a potential environmental risk factor contributing to epileptogenesis.甲基汞：可能导致癫痫发生的环境风险因素。

Neurotoxicology. 2012 Jan;33(1):119-26. doi: 10.1016/j.neuro.2011.12.014. Epub 2011 Dec 22.

In vivo methylmercury exposure induced long-lasting epileptiform activity in layer II/III neurons in cortical slices from the rat.体内甲基汞暴露可在大鼠皮质切片的 II/III 层神经元中诱导持久的癫痫样活性。

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Does methylmercury have a role in causing developmental disabilities in children?甲基汞在导致儿童发育障碍方面起作用吗？

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Environmental release of chemicals and reproductive ecology.化学物质的环境释放与生殖生态学。

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