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Micropuncture study of the proximal tubular factors responsible for the maintenance of alkalosis during potassium deficiency in the rat.

作者信息

Kunau R T, Frick A, Rector F C, Seldin D W

出版信息

Clin Sci. 1968 Apr;34(2):223-31.

PMID:5653684
Abstract
摘要

相似文献

1
Micropuncture study of the proximal tubular factors responsible for the maintenance of alkalosis during potassium deficiency in the rat.对大鼠缺钾期间维持碱中毒的近端小管因素的微穿刺研究。
Clin Sci. 1968 Apr;34(2):223-31.
2
A micropuncture study of renal bicarbonate and chloride reabsorption in hypokalaemic alkalosis.低钾血症性碱中毒时肾脏对碳酸氢根和氯离子重吸收的微穿刺研究
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3
Does chloride play an independent role in the pathogenesis of metabolic alkalosis?
Semin Nephrol. 1988 Jun;8(2):101-8.
4
Induction of metabolic alkalosis by the correction of potassium deficiency.通过纠正钾缺乏诱导代谢性碱中毒。
Trans Assoc Am Physicians. 1965;78:82-9.
5
Potassium transport across renal distal tubules during acid-base disturbances.酸碱平衡紊乱时钾离子在肾脏远曲小管的转运
Am J Physiol. 1971 Oct;221(4):1192-208. doi: 10.1152/ajplegacy.1971.221.4.1192.
6
The induction of metabolic alkalosis by correction of potassium deficiency.通过纠正钾缺乏症诱发代谢性碱中毒。
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7
Renal tubular microinjection studies in normal and potassium-depleted rats.正常及低钾大鼠的肾小管微量注射研究。
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8
In vivo evidence of impaired solute transport by the thick ascending limb in potassium-depleted rats.钾缺乏大鼠厚壁升支溶质转运受损的体内证据。
J Clin Invest. 1984 Apr;73(4):908-16. doi: 10.1172/JCI111314.
9
Potassium-depletion alkalosis in the rat.大鼠的低钾性碱中毒
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10
[Metabolic alkalosis].[代谢性碱中毒]
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引用本文的文献

1
NBCe1-A is required for the renal ammonia and K response to hypokalemia.NBCe1-A 对于低钾血症时肾脏氨和钾的反应是必需的。
Am J Physiol Renal Physiol. 2020 Feb 1;318(2):F402-F421. doi: 10.1152/ajprenal.00481.2019. Epub 2019 Dec 16.
2
Chloride/bicarbonate exchanger SLC26A7 is localized in endosomes in medullary collecting duct cells and is targeted to the basolateral membrane in hypertonicity and potassium depletion.氯离子/碳酸氢根离子交换体SLC26A7定位于髓质集合管细胞的内体中，并在高渗和钾缺乏时靶向至基底外侧膜。
J Am Soc Nephrol. 2006 Apr;17(4):956-67. doi: 10.1681/ASN.2005111174. Epub 2006 Mar 8.
3
Transepithelial electrochemical gradients in the proximal convoluted tubule during potassium depletion in the rat.
大鼠钾缺乏时近端曲管中的跨上皮电化学梯度
J Physiol. 1998 Dec 1;513 ( Pt 2)(Pt 2):551-7. doi: 10.1111/j.1469-7793.1998.551bb.x.
4
Diuretics stimulate H+ secretion in turtle urinary bladder.利尿剂刺激龟膀胱中的氢离子分泌。
J Clin Invest. 1980 May;65(5):1095-103. doi: 10.1172/JCI109762.
5
Metabolic alkalosis in the rat. Evidence that reduced glomerular filtration rather than enhanced tubular bicarbonate reabsorption is responsible for maintaining the alkalotic state.大鼠的代谢性碱中毒。有证据表明，维持碱中毒状态的原因是肾小球滤过率降低，而非肾小管对碳酸氢盐的重吸收增强。
J Clin Invest. 1983 May;71(5):1141-60. doi: 10.1172/jci110864.
6
Volume-independent reductions in glomerular filtration rate in acute chloride-depletion alkalosis in the rat. Evidence for mediation by tubuloglomerular feedback.大鼠急性氯耗竭性碱中毒时肾小球滤过率的容量非依赖性降低。管球反馈介导的证据。
J Clin Invest. 1984 Dec;74(6):2002-8. doi: 10.1172/JCI111622.
7
Bicarbonate reabsorption in the papillary collecting duct of rats.大鼠乳头集合管中的碳酸氢盐重吸收
Pflugers Arch. 1981 Mar;389(3):271-5. doi: 10.1007/BF00584789.
8
Proximal tubular bicarbonate reabsorption and PCO2 in chronic metabolic alkalosis in the rat.大鼠慢性代谢性碱中毒时近端肾小管碳酸氢盐重吸收与二氧化碳分压
J Clin Invest. 1983 Oct;72(4):1385-95. doi: 10.1172/JCI111095.
9
The influence of the carbonic anhydrase inhibitor, benzolamide (CL-11,366), on the reabsorption of chloride, sodium, and bicarbonate in the proximal tubule of the rat.碳酸酐酶抑制剂苯佐拉胺（CL-11,366）对大鼠近端小管中氯离子、钠离子和碳酸氢根重吸收的影响。
J Clin Invest. 1972 Feb;51(2):294-306. doi: 10.1172/JCI106814.
10
Potassium depletion increases luminal Na+/H+ exchange and basolateral Na+:CO3=:HCO3- cotransport in rat renal cortex.钾缺乏会增加大鼠肾皮质管腔的Na+/H+交换以及基底外侧的Na+:CO3=:HCO3-协同转运。
J Clin Invest. 1990 Oct;86(4):1076-83. doi: 10.1172/JCI114810.