Bettini V, Legrenzi E, Vazzoler C, Malgherini A
Boll Soc Ital Biol Sper. 1978 Sep 15;54(17):1642-6.
The mechanical responses of coronary arteries to Ach were studied in vitro. Ach caused always a contraction of the preparation, which was prevented or abolished by Athropine but not by Exametonium. Therefore Ach seems to act through muscarinic receptors.
在体外研究了冠状动脉对乙酰胆碱(Ach)的机械反应。乙酰胆碱总是引起标本收缩,阿托品可阻止或消除这种收缩,但依酚氯铵不能。因此,乙酰胆碱似乎是通过毒蕈碱受体起作用的。